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Text-mined interactions from Literome

Wei et al., J Biol Chem 2002 (MAP Kinase Signaling System) : We find that although pretreatment of BMMs with IL-4 does not alter M-CSF signaling, it reversibly blocks RANKL dependent activation of the NF-kappa B, JNK, p38 , and ERK signals
Lee et al., Bone 2002 : RANKL also stimulated the activity of p38
Ishida et al., J Biol Chem 2002 : Therefore, protein kinase A and p38 signaling pathways are involved in TGF-beta induced RANKL expression by stimulating transcription factors that bind to the CRE-like domains
Wang et al., J Bone Miner Res 2003 : Assays for NF-kappaB nuclear translocation, NF-kappaB reporter gene activity, protein kinase activity, and Western blotting were used to examine the effects of TPA on RANKL induced NF-kappaB, c-Jun N-terminal kinase (JNK), and MEK/ERK and p38 signal transduction pathways
Fujimura et al., Infect Immun 2006 : RANKL induced phosphorylation of ERK, p38 , and NF-kappaB was not suppressed by HbR, but that of Akt was markedly suppressed
Mori et al., Bone 2007 (Bone Neoplasms...) : Indeed, soluble human RANKL ( shRANKL, 100 ng/ml ) treatment induced ERK 1/2, p38 and IkappaB phosphorylations in these cells
Mozar et al., J Cell Physiol 2008 (Bone Resorption) : Pi was found to specifically inhibit the RANKL induced JNK and Akt activation, while RANKL induced p38 and ERK 1/2 phosphorylation were not significantly affected
Mazière et al., J Cell Physiol 2009 (Bone Resorption) : OxLDL also prevented the RANKL induced phosphorylation of ERK, p38 and JNK kinases, together with the RANKL induced DNA binding activities of NFkappaB and NFAT transcription factors
Kim et al., Anatomy & cell biology 2010 : Amorphigenin markedly inhibited RANKL induced p38 and NF-?B pathways, but other pathways were not affected
Liu et al., Int Immunopharmacol 2012 (Bone Resorption...) : In addition, D-pinitol also reduced RANKL induced p38 and JNK phosphorylation ... Our data suggest that D-pinitol inhibits osteoclastogenesis from bone marrow stromal cells and macrophage cells via attenuated RANKL induced p38 , JNK, and NF-?B activation, which in turn protect bone loss from ovariectomy
Moon et al., Phytother Res 2012 : The SSE thereafter suppressed RANKL induced p38 mitogen activated protein kinase and I?Ba kinase signalling activities which were activated by ROS generation for osteoclastogenesis
Ko et al., Arch Oral Biol 2012 : Myricetin also inhibited the RANKL stimulated activation of p-38 , ERK and cSrc signaling, and inhibited the RANKL stimulated degradation of I ( k ) B in the RAW264.7 cells
Kim et al., Life Sci 2012 (Bone Resorption) : NecroX-7 significantly inhibited the NF-?B signaling pathway without affecting the activation of the mitogen activated protein kinases ( MAPKs ) JNK, p38 , and ERK in response to RANKL