◀ Back to NOX4

APOA1 — NOX4

Text-mined interactions from Literome

Tabet et al., Arterioscler Thromb Vasc Biol 2011 (Diabetes Mellitus, Type 2) : Lipid-free apoA-I and (A-I)rHDL increased SOD1 and SOD2 levels and attenuated 25 mmol/L glucose mediated increases in cellular O2-, NADPH oxidase activity, p47 translocation, and Nox2 expression ... Lipid-free apoA-I mediated its effects on Nox2 , SOD1, and SOD2 via ABCA1
Umemoto et al., Circ Res 2013 (Disease Models, Animal...) : ApoAI and HDL also disrupted LRs, reduced plasma membrane cholesterol content, inhibited NADPH oxidase 4 translocation into LRs, and reduced palmitate induced reactive oxygen species generation and monocyte chemotactic factor expression