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NOS3 — SOD1

Text-mined interactions from Literome

Johnson et al., J Appl Physiol 2001 : Immunoblot analysis indicated a 3.9-fold increase in eNOS protein in pulmonary artery tissue from STEx pigs ( P < 0.05 ) with no change in SOD-1 or glyceraldehyde-3-phosphate dehydrogenase protein levels
Vouldoukis et al., Mol Med 2000 : The concomitant stimulation of the FcyR and the translocation of the SOD1 in the cytoplasm of IFN-gamma activated macrophages not only reduced the production of superoxide anion but also induced the expression of the inducible form of nitric oxide synthase ( iNOS ) and the related NO production
Brennan et al., IUBMB Life 2002 (Hypertension) : Previously, we have demonstrated that increased superoxide generation plays a role in the nitric oxide ( NO ) -mediated inhibition of endothelial NO synthase (eNOS) in endothelial cells ( ECs ) and that the overexpression of SOD1 could reduce the inhibitory effect of NO
Rojas et al., Exp Physiol 2003 : Incubation with D-glucose activated protein kinase C ( PKC ), endothelial NO synthase (eNOS) , p42 and p44 mitogen activated protein kinases ( p42/44(mapk) ), but inhibited superoxide dismutase ( SOD )
Polytarchou et al., Eur J Pharmacol 2005 : Superoxide dismutase ( SOD ) , tempol ( membrane permeable SOD mimetic ) and the NADPH oxidase inhibitors, 4- ( 2-aminoethyl ) -benzenesulfonyl fluoride and apocynin, but not allopurinol, inhibited HUVEC proliferation and migration, as well as activity of endothelial NOS (eNOS)
Lee et al., Exp Mol Med 2009 : Differential regulation of inducible nitric oxide synthase and cyclooxygenase-2 expression by superoxide dismutase in lipopolysaccharide stimulated RAW 264.7 cells
Choi et al., Cell Physiol Biochem 2010 : LPC concentration- and time-dependently decreased eNOS levels, but this effect was blocked by antioxidants and SOD and potentiated by the SOD1 inhibitor, ammonium tetrathiomolybdate
Afolayan et al., Am J Physiol Lung Cell Mol Physiol 2012 (Disease Models, Animal...) : We investigated the hypothesis that a decrease in expression and activity of mitochondrial localized manganese superoxide dismutase ( MnSOD ) in pulmonary artery endothelial cells ( PAEC ) increases oxidative stress and impairs eNOS function in PPHN
Berman et al., Br J Pharmacol 1993 : 3. The increase in albumin transfer induced by hypoxanthine and xanthine oxidase was abolished by catalase ( 3 u ml-1 ), reduced by allopurinol ( 4 mM ), but unaffected by superoxide dismutase ( 6000 u ml-1 ), the hydroxyl radical scavengers, mannitol ( 15 mM ), dimethylthiourea ( 10 mM ) and N- ( 2-mercaptopropionyl ) -glycine ( 1 mM ), the iron chelator, deferoxamine ( 0.5 mM ), ferric chloride ( 50 microM ), an inhibitor of nitric oxide synthase , NG-nitro-L-arginine ( 30 microM ), or the antioxidant, dithiothreitol ( 3 mM )
Allen et al., J Infect Dis 1997 : The possibility that peroxynitrite contributes to this maximum luminol dependent activity was tested, but neither superoxide dismutase , a competitive inhibitor of peroxynitrite production, nor N-methyl-L-arginine, an inhibitor of nitric oxide synthase , exerted a significant inhibitory effect
Nelson et al., Hum Reprod 1998 : In both P and NP arteries the cholinergic relaxation was increased in the presence of superoxide dismutase and greatly reduced in the presence of the nitric oxide synthase inhibitors, NG-mono-methyl L-arginine ( L-NMMA ) and L-nitro-arginine-methylester ( L-NAME )