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FCER2 — TNF

Text-mined interactions from Literome

Hunot et al., J Neurosci 1999 (Parkinson Disease) : The activation of CD23 also led to an upregulation of TNF-alpha production, which was dependent on NO release
Chabot et al., J Immunol 1999 : By also measuring TNF-alpha levels, specificity of cytokine regulation was observed ; while anti-CD40 and CTLA-4-Fc reduced IL-10 and TNF-alpha levels, anti-CD23 did not affect TNF-alpha while attenuating IL-10 generation
Mitsuta et al., Allergy Asthma Proc 2003 (Asthma...) : Our results indicated that TNF-alpha production was induced by D. farinae in PBMCs of patients with atopic asthma by the activation of NF-kappa B via CD23
Harkins et al., Ann Allergy Asthma Immunol 2003 (Asthma...) : Stimulating the CD23 receptor with immune complexes significantly increased TNF-alpha production by THP-1 cells stimulated with IFN-gamma, IL-4, IL-13, or a combination of these
Morita et al., Blood 1991 (Leukemia, Eosinophilic, Acute...) : However, G-CSF and TNF-alpha did not induce the expression of Fc epsilon RII and EO-1 antigen
Mossalayi et al., Infect Immun 2009 : CD23 activation also induced tumor necrosis factor alpha (TNF-alpha) production from MDM. Mycobacteria reduction was partially inhibited by the addition of neutralizing anti-TNF-alpha antibody to cell cultures without affecting NO levels, which suggested the role of this cytokine for optimal antimicrobial activity
Tu et al., North American journal of medical sciences 2009 : Nuclear factor kappa B inhibitor, tryptase and TNF-a inhibited the increase in CD23 in HT29 cells and prevents the enhancement of epithelial barrier permeability
Kwon et al., J Cell Sci 2012 : We found that downregulation of CD23 attenuated AIMP1 induced TNF-a secretion and AIMP1 binding to THP-1 and PBMCs ... We also observed that in THP-1 and PBMCs, AIMP1 induced TNF-a secretion, mediated by CD23 , involved activation of ERK1/2
Bécherel et al., Cell Mol Biol Incl Cyto Enzymol 1994 : In the present study, we showed that the ligation of CD23 by IgE/anti-IgE immune complexes or specific monoclonal antibody, induces a dose dependent release of interleukin-6 and tumor necrosis factor-alpha from EK
Boltz-Nitulescu et al., J Leukoc Biol 1995 : Fc epsilon RII was also triggered by IFN-gamma, TNF-alpha , and IL-6 on all the cell lines, an effect blocked by calcitriol
Hart et al., J Immunol 1993 (Arthritis...) : Activation and differentiation in vitro of blood monocytes reduced their response to IL-4 for decreased TNF-alpha production and induction of CD23 and suggested that these biologic phenomena may contribute to the decreased responsiveness to IL-4 by synovial fluid monocytes/macrophages
Ouaaz et al., Blood 1994 (Leukemia, Myelomonocytic, Acute) : CD23 ligation also induced the production of TNF alpha by U937 cells
Bécherel et al., J Clin Invest 1994 : In the present study, we showed that the ligation of CD23 by IgE/anti-IgE immune complexes or specific monoclonal antibody induces a dose dependent release of interleukin 6 and tumor necrosis factor-alpha from EK
Gessl et al., Immunology 1993 : Interestingly, on human monocytes TNF-alpha weakly reduced the basal level of Fc epsilon RII , and markedly diminished the IL-4 induced Fc epsilon RII expression
Dugas et al., Eur J Immunol 1996 : In the presence of Rp-cAMP, the CD23 induced production of IL-10 and of the pro-inflammatory cytokine tumor necrosis factor-alpha (TNF-alpha) was totally abrogated, whereas, in the presence of L-NMMA, IL-10 production was enhanced and TNF-alpha production was suppressed
Bécherel et al., J Invest Dermatol 1996 : Immunoglobulin E ligates CD23 and induces the production of nitrites ( reflecting the mobilization of the nitric oxide [ NO ] -pathway ) and tumor necrosis factor-alpha by human keratinocytes
Ouaaz et al., Mol Med 1996 : p24 induction by IgE-IC was then shown to be due to CD23 mediated stimulation of cAMP, NO, and tumor necrosis factor alpha (TNF alpha) generation
Hashimoto et al., Allergy 1997 : N-acetylcysteine attenuates TNF-alpha dependent reduction of IL-4 induced Fc epsilon RII expression in human monocytes ... We have previously shown that tumor necrosis factor-alpha (TNF-alpha) reduces interleukin-4 (IL-4) induced Fc epsilon RII expression in human monocytes ... Therefore, we hypothesized that TNF-alpha dependent reduction of IL-4 induced Fc epsilon RII expression in monocytes might be mediated through the ROIs activated mechanism ... In the present study, to test our hypothesis, we examined the effect of NAC on TNF-alpha dependent reduction of IL-4 induced Fc epsilon RII expression in human monocytes ... NAC attenuated TNF-alpha dependent reduction of IL-4 induced Fc epsilon RII expression by attenuating TNF-alpha dependent reduction of Fc epsilon RII mRNA expression ... These results indicate that an ROIs activated and antioxidant-sensitive mechanism might be involved in TNF-alpha dependent reduction of IL-4 induced Fc epsilon RII expression in monocytes
Bécherel et al., J Immunol 1997 : Treatment of HK with anti-IL-10 mAb potentiated their CD23 mediated TNF-alpha synthesis