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HMOX1 — IFNG

Text-mined interactions from Literome

Kitamura et al., Neurosci Lett 1999 : However, PPARgamma activator alone induced HO-1 expression and further enhanced LPS/IFN-gamma induced HO-1 expression
Datta et al., Kidney Int 1999 : HO-1 expression was also increased in response to LPS/IFN-gamma
Takahashi et al., J Neurochem 1999 (Glioblastoma) : These findings raise the possibility that the expression of heme oxygenase-1 is down-regulated by interferon-gamma in the nervous system
Chen et al., J Cell Biochem 2002 : Nitric oxide and prostaglandin E2 participate in lipopolysaccharide/interferon-gamma induced heme oxygenase 1 and prevent RAW264.7 macrophages from UV-irradiation induced cell death
Kim et al., Exp Mol Med 2003 (Urinary Bladder Neoplasms) : Simultaneously, TMAF induced HO-1 and this induction was slightly augmented by IFN-gamma
Udono-Fujimori et al., Eur J Biochem 2004 : Expression of heme oxygenase-1 is repressed by interferon-gamma and induced by hypoxia in human retinal pigment epithelial cells
Malaguarnera et al., Int Immunopharmacol 2005 : Moreover, co-stimulation of PRL with LPS caused activation of HMMs functions, enhancement of HO-1 expression and induction of VEGF release, whereas addition of PRL inhibited up-regulation of HO-1 or VEGF induced by IFN-gamma or TNF-alpha
Wood et al., Trends Immunol 2006 : Furthermore, it can indirectly prevent further T-cell activation by creating a microenvironment that influences the function of antigen presenting cells (APCs) as a result of IFNgamma induced inducible nitric oxide synthase (iNOS), indoleamine-2,3-dioxygenase (IDO) and heme oxygenase (HO)-1 expression
Lehmann et al., J Invest Dermatol 2007 : Moreover, inhibition of HO-1 activity restored the diminished IL-12 and IFNgamma production after FAE treatment
Cudmore et al., Circulation 2007 (Pre-Eclampsia) : Adenoviral overexpression of HO-1 in endothelial cells inhibited VEGF mediated sFlt-1 release and interferon-gamma- and tumor necrosis factor-alpha induced sEng release, whereas HO-1 inhibition potentiated sFlt-1 and sEng production from endothelial cells and placental villous explants