Zhou et al., Mol Endocrinol 1999
:
Nevertheless, as demonstrated in this study, both IRS-3 and IRS-4 can also stimulate translocation of GLUT4 ... Interestingly, overexpression of IRS-3-F4 did not promote translocation of GLUT4-HA , but actually inhibited the ability of insulin to stimulate translocation of GLUT4-HA to the cell surface
Xu et al., Eur Rev Med Pharmacol Sci 2011
(Diabetes Mellitus, Experimental) :
Effect of insulin in combination with selenium on Irs/PI3K mediated GLUT4 expression in cardiac muscle of diabetic rats