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CA2 — DHPS

Text-mined interactions from Literome

Chesnoy-Marchais et al., Eur J Neurosci 2001 (Synaptic Transmission) : At concentrations between 1 and 10 microM, all these DHPs could block glycine responses, even in the absence of extracellular Ca2+
Villalobos et al., Pflugers Arch 1995 : Any of the above- described treatments induced a sustained increase in intracellular Ca2+ concentration ([Ca2+]i) , which was dependent on extracellular Ca2+ and was prevented by Ni2+ but not by dihydropyridines (DHPs) , suggesting that it was due to capacitative Ca2+ entry via activation of a plasma membrane pathway which opened upon the emptying of the intracellular Ca2+ stores
Tanaka et al., J Physiol 1997 (Ion Channel Gating) : To distinguish maxi KCa currents modulated by the beta subunit, we examined ( a ) their apparent Ca2+ sensitivity, as judged from the voltage necessary to half-activate the channel ( V1/2 ), and ( b ) their activation by DHS-I