◀ Back to HNRNPF

AGT — HNRNPF

Text-mined interactions from Literome

Lo et al., Diabetes 2012 (Diabetes Mellitus, Experimental...) : The results showed that hnRNP F overexpression attenuated systemic hypertension, suppressed Agt and transforming growth factor-ß1 ( TGF-ß1 ) gene expression, and reduced urinary Agt and angiotensin II levels, renal hypertrophy, and glomerulotubular fibrosis in Akita hnRNP F-Tg mice ... In vitro, hnRNP F overexpression prevented the high-glucose stimulation of Agt and TGF-ß1 mRNA expression and cellular hypertrophy in RPTCs
Abdo et al., Diabetologia 2013 : In vitro, insulin inhibited Agt but stimulated Hnrnpf and Hnrnpk expression in high-glucose media via p44/42 mitogen activated protein kinase signalling in RPTCs. Transfection with Hnrnpf or Hnrnpk small interfering RNAs prevented insulin inhibition of Agt expression in RPTCs