◀ Back to PCNA

CCNG1 — PCNA

Text-mined interactions from Literome

Bouchard et al., J Virol 2001 : Instead, HBx is shown to activate the cyclin A promoter, induce cyclin A-cyclin dependent kinase 2 complexes, and promote cycling of growth arrested cells into G1 through a pathway involving activation of Src tyrosine kinases
Piscopo et al., Cancer Res 2008 : Ectopic cyclin G1 protein stability is increased by cyclin box mutation or by association with inactive cyclin dependent kinase (CDK) subunits, suggesting that a function of cyclin G1 as a CDK regulator may be required for its rapid turnover
Santo et al., Oncogene 2010 (Multiple Myeloma) : Dysregulated cell cycling is a universal hallmark of cancer and is often mediated by abnormal activation of cyclin dependent kinases (CDKs) and their cyclin partners
Soffar et al., Int J Radiat Biol 2013 (Head and Neck Neoplasms) : Abstract Purpose : Cyclin dependent kinase 2 (CDK2) is critically involved in cell cycling and has been proposed as a potential cancer target
Elias et al., Int J Dermatol 1995 (Psoriasis) : Since PCNA is a marker of cellular proliferation and p53 inhibits cellular cycling , some of the beneficial effects of PTU and MMI in psoriasis may depend on the ability of the drugs to impair cellular turnover, perhaps by binding to the triiodothyronine ( T3 ) receptor