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EDN3 — NOS2

Text-mined interactions from Literome

Barton et al., J Am Soc Nephrol 2000 (Glomerulosclerosis, Focal Segmental...) : Dysfunctional renal nitric oxide synthase as a determinant of salt-sensitive hypertension : mechanisms of renal artery endothelial dysfunction and role of endothelin for vascular hypertrophy and Glomerulosclerosis
Mérial-Kieny et al., Br J Pharmacol 2003 : 3. ET-1, but not ET-3 , inhibited the TNFalpha induced expression of iNOS protein and mRNA as well as nitrite production
Sudjarwo et al., Eur J Pharmacol 1992 : These results suggest that ET-3 acts on ETB receptors in the vascular endothelium to increase [Ca2+ ] i by releasing Ca2+ from storage sites and by opening non-L type Ca2+ channels, activates nitric oxide synthase , releases nitric oxide and relaxes vascular smooth muscle
Ohlstein et al., J Neurosurg 1992 : An l-arginine analog inhibitor of nitric oxide synthase , L-NG-monomethyl arginine ( L-NMMA, 200 microM ), did not significantly affect basal immunoreactive endothelin levels
Novella et al., Eur J Cardiothorac Surg 2007 (Cardiovascular Diseases) : No change in the AVP response was produced by endothelium removal, exposure to the NO precursor ( 3 x 10 ( -4 ) M L-arginine ), inhibition of nitric oxide ( NO ) synthase ( 3 x 10 ( -5 ) M L-NAME ) or soluble guanylate cyclase ( 3 x 10 ( -6 ) M 1H- [ 1,2,4 ] oxadiazol [ 4,3,-alpha ] quinoxalin-1-one ( ODQ ) ), removal of the superoxide anion ( 100 U/ml superoxide dismutase ( SOD ) plus 1200 U/ml catalase ) or hydroxyl radical ( 10 ( -4 ) M deferoxamine ), or specific alpha1 - ( 10 ( -6 ) M prazosin ) or endothelin ( 10 ( -5 ) M bosentan ) receptor antagonism
Chin et al., J Periodontal Res 2009 (Mouth, Edentulous) : The roles of the endothelin receptors A and B ( ET ( A ) and ET ( B ) ) in CsA enhanced expression of PCNA and iNOS were examined in cultured human gingival fibroblasts pretreated with receptor antagonists, by immunocytochemistry and RT-PCR, respectively
Hirahashi et al., Pharmacology 1996 : To investigate the interaction between endothelin ( ET ) and the nitric oxide system, we examined the effects of ET-1 and ET-3 on the induction of inducible nitric oxide synthase (iNOS) and guanosine triphosphate cyclohydrolase I ( GTP:CHI ), the rate limiting enzyme of de novo synthesis of the cofactor tetrahydrobiopterin ( BH4 ), in rat mesangial cells
Oda et al., Eur J Pharmacol 1997 : We found that in glial cell cultures lipopolysaccharide stimulated inducible NO synthase expression and nitrite accumulation were synergistically enhanced by pretreatment with endothelin, whereas endothelin itself did not induce these responses
Lin et al., Eur J Endocrinol 1998 : Role of nitric oxide synthase in release of endothelin from cultured human endometrial cells
Murayama et al., Life Sci 1998 : Regulation of inducible NO synthase expression by endothelin in primary cultured glial cells