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CD69 — IL2

Text-mined interactions from Literome

Lin et al., Biol Neonate 2000 : The effect of interleukin-12 and interleukin-15 on CD69 expression of T-lymphocytes and natural killer cells from umbilical cord blood
Gan et al., Brain Behav Immun 2002 : We also demonstrate that NE inhibited the IL-2 mediated up-regulation of the activation marker CD69
Luxembourg et al., Cell Immunol 2002 : Up-regulation of CD25 and CD69 and proliferation were less efficiently induced ( 20-30-fold more sensitive ), and interleukin (IL)-2 production was induced least efficiently ( only 2-fold more sensitive )
Tchórzewski et al., Med Sci Monit 2005 (Autoimmune Diseases...) : We analyzed reactive oxygen intermediate ( ROI ) production by peripheral blood granulocytes, the production of IL-2 , IL-5, IL-10, IL-12, and TNF-alpha cytokines by lymphocytes, and the activation of CD25, CD26, CD69 , CD71, and HLA DR antigen expression
Yu et al., Zhong Yao Cai 2005 : the immunosuppressive mechanism of TRD may be related to the inhibitory effects on IL-2 and IFN-gamma mRNA expression and the inhibition of CD69 and CD25 expression
Zhao et al., Leukemia 2008 (Hypersensitivity, Delayed) : sorafenib also caused G ( 0 ) /G ( 1 ) phase arrest, inhibition of CD25 and CD69 expression, interleukin-2 production and LCK phosphorylation in the T cells ; all of these effects exhibited dose and time dependence ... sorafenib also caused G ( 0 ) /G ( 1 ) phase arrest, inhibition of CD25 and CD69 expression, interleukin-2 production and LCK phosphorylation in the T cells ; all of these effects exhibited dose and time dependence
Weichsel et al., Clin Cancer Res 2008 : Functional outcomes included cytokine production ( interleukin-2 , IFN gamma, and tumor necrosis factor alpha ), degranulation ( CD107a/b mobilization ), activation ( CD69 up-regulation ), proliferation ( carboxyfluorescein diacetate succinimidyl ester dilution ), apoptosis/necrosis induction, and signal transduction
Nerreter et al., Exp Hematol 2013 : Functional outcomes assessed included cytokine production ( IL-2 , IFN-?, TNF-a ), degranulation ( CD107a/b ), activation ( CD69 upregulation ), proliferation, apoptosis and necrosis induction, and signal transduction
Testi et al., J Immunol 1989 : When PKC was simultaneously activated by PMA, stimulation of CD69 induced IL-2 and IFN-gamma gene expression, enhancement of CD25 expression, and ultimately IL-2 dependent T cell proliferation ... Cyclosporin A, which does not prevent PKC induced CD69 expression, completely suppressed CD69 induced IL-2 and IFN-gamma gene expression
Vanham et al., Clin Immunol Immunopathol 1994 (HIV Infections...) : To evaluate the function of IL2R, we investigated to what extent IL2 could induce CD69 , an early activation marker of lymphocytes ... Based on inhibition studies, both the alpha and the beta chain contributed to the IL-2 induced CD69 expression on CD4 T cells, pointing to involvement of the high-affinity receptor
Shenker et al., J Leukoc Biol 1994 : LTX inhibited the IL-2 induced expression of both CD69 and the IL-2 receptor
Kjeldsen-Kragh et al., Eur J Immunol 1993 : Finally, we found that the IL-2 induced expression of CD69 was inhibited by activation of cAMP dependent protein kinase and by inhibition of the Src-family of the tyrosine protein kinase, but not by inhibition of protein kinase C or by activation of the CD45 associated tyrosine phosphatase
Borrego et al., Eur J Immunol 1993 : We found that CD69 is induced on NK cells not only by IL-2 and IFN-alpha but also by activation of the CD16 pathway, the interaction with NK target cells and the direct activation of PKC by phorbol 12-myristate 13-acetate ( PMA ), indicating that CD69 induction is associated to different NK activation pathways ... However, it did not significantly affect CD69 induction by IL-2 , IFN-alpha or CD16 cross linking ... On the other hand genistein, a competitive inhibitor of PTK enzymes, blocked the expression of CD69 induced by activation of NK cells via IL-2 or IFN-alpha receptors, CD16 and K562 receptor ( s ), indicating that stimulation of PTK is a common step in the signal transduction events leading to the induction of CD69 antigens after the activation of NK cells via these receptors
Lim et al., Mol Cells 1998 : They were also capable of stimulating IL-2 gene expression in a NF-AT dependent manner and inducing CD69 expression on the surface