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ICAM1 — IFNA2
Text-mined interactions from Literome
Håkansson et al., J Interferon Cytokine Res 1999
(Melanoma) :
We used monoclonal antibodies, anti-ICAM-1, anti-CD4+, anti-CD8+, and anti-CD11c+ to study the
effect of
interferon-alpha ( IFN-alpha ) on the expression of
ICAM-1 by melanoma cells in regional metastases and its correlation to the occurrence of CD4+, CD8+, and CD11c+ cells close to tumor cells and in the tumor stroma
Foster et al., J Immunol 2004
:
In this study, we show that
IFN-alpha 2 induces chemokinesis of both CD4 ( + ) and CD8 ( + ) T cells at various stages of differentiation, and induces functional changes that result in enhanced T cell motility, including up-regulation of the integrins LFA-1 and VLA-4, and subsequently,
increased ICAM-1- and fibronectin dependent migration
Böhm et al., Exp Dermatol 2004
:
Functional studies have shown that alpha-MSH exerts anti-inflammatory actions in human fibroblastic skin cells by suppressing interleukin-1 (IL-1) induced IL-8 production, activation of the transcription factor activator protein-1 (AP-1) and
induction of
intercellular adhesion molecule-1 by
interferon-alpha
Krutmann et al., J Invest Dermatol 1990
:
Stimulation of cells with recombinant human ( rh ) interleukin (IL) 1 alpha, rhIL-4, rhIL-5, rhIL-6, rh granulocyte/macrophage colony stimulating factor ( GM-CSF ), rh
interferon alpha ( rhIFN alpha ), and rh transforming growth factor beta ( TGF beta ) did not
increase ICAM-1 surface expression
Campbell et al., J Urol 1994
(Carcinoma, Transitional Cell...) :
In contrast, interleukin-1 and phorbol myristate acetate exhibited variable effects on
ICAM-1 expression, and
interferon-alpha had no effect