◀ Back to EPHB2

EPHB2 — NTF3

Text-mined interactions from Literome

Yuen et al., Exp Neurol 1999 : BDNF, NT-3 , and NT-4 caused rapid tyrosine phosphorylation of ERK and SNT
Bron et al., Mol Cell Neurosci 2003 (Pain) : Dominant negative Ras, but not Rap, blocks NTF induced ERK activation and VR1 upregulation ... However, this may at least in part be due to a block of NTF induced ERK activation
Gozdz et al., J Biol Chem 2003 : Finally, CPDD toxicity was reduced by brain derived neurotrophic factor , and this protection required ERK1/2
Althini et al., Mol Cell Neurosci 2004 (MAP Kinase Signaling System) : Phosphorylation of Erk2 , induced by NT3 , was reduced by MEK inhibition but unaffected by BMP signaling
Sturla et al., Cancer Res 2005 (Cerebellar Neoplasms...) : Co-immunoprecipitation studies revealed direct interaction of phosphorylated ERK5 with MEF2 in response to neurotrophin-3
Shinoda et al., J Neurosci 2007 : These results suggest that DISC1 is required for NT-3 induced axon elongation and ERK activation at the distal part of axons by recruiting Grb2 to axonal tips
Wilson-Gerwing et al., J Comp Neurol 2009 (MAP Kinase Signaling System) : NT-3 did not significantly attenuate levels of phospho-ERK1/2 when p75NTR expression was suppressed by antisense infusion, despite being able to do so when NT-3 was infused alone
Moubarak et al., J Neurosci 2010 : Altogether, we uncover a new role for FLIP-L as an unexpected critical player in neurotrophin induced mitogen activated protein kinase/ERK- and NF-kappaB mediated control of neurite growth in developing neurons
Kommaddi et al., FASEB J 2011 : We show that Trk induced ADAM17 phosphorylation and generation of the p75NTR ( ICD ) is required for neurotrophin induced Erk and Akt activation and for neurotrophin dependent survival signaling
Hwang et al., J Agric Food Chem 2011 : Hesperetin also stimulated the activation of Akt, ERK , and CREB as well as induced brain derived neurotrophic factor , PPAR? coactivator 1a ( PGC-1a ), and seladin-1 ( selective Alzheimer 's disease indicator-1 ) via both ER and TrkA in the cells
Li et al., Toxicological sciences : an official journal of the Society of Toxicology 2013 : 6-OH-PBDE-47 also inhibited the proneuronal differentiation effect of neurotrophin 3 (NT3) and NT3 activation of ERK5