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ICAM3 — TNF

Text-mined interactions from Literome

Bleijs et al., Eur J Immunol 1999 : In contrast, ICAM-2 and ICAM-3 induced a much stronger secretion of the Th1 cytokine TNF-alpha compared to LFA-1/ICAM-1 induced co-stimulation, despite the lower proliferation rate ... In contrast, ICAM-2 and ICAM-3 induced a much stronger secretion of the Th1 cytokine TNF-alpha compared to LFA-1/ICAM-1 induced co-stimulation, despite the lower proliferation rate
Walther et al., Int J Radiat Biol 1999 : Treatment with NAC clearly restrained TNF-alpha induced ICAM expression on HUVEC, while preincubation of cells with PDTC showed synergistic effects
De Caterina et al., Am J Clin Nutr 2000 (Arteriosclerosis) : Consumption of the n-3 fatty acid docosahexaenoic acid ( DHA ; 22 : 6n-3 ) reduced endothelial expression of vascular cell adhesion molecule 1 ( VCAM-1 ), E-selectin, intercellular adhesion molecule 1 ( ICAM-1 ), interleukin 6 (IL-6), and IL-8 in response to IL-1, IL-4, tumor necrosis factor , or bacterial endotoxin, with a half-maximal inhibitory concentration ( IC ( 50 ) ) of 1-25 micromol, ie, in the range of nutritionally achievable plasma concentrations
Furukawa et al., J Vasc Surg 2000 : Finally, the specificity of the effect of transduction of ICP47 on vascular cell MHC-I expression was confirmed by a lack of significant change in either constitutive or tumor necrosis factor induced vascular cell adhesion molecule/intercellular adhesion molecule expression
Gnant et al., Cytokine 2000 : TNF caused increased secretion of IL-6, IL-8, ICAM-1 , and VCAM-1 with little or no additional effect from HT. Increased E-selectin and RANTES levels were observed with TNF and HT only at 24 h after treatment
Grünberg et al., Tumour Biol 2000 (Breast Neoplasms) : Furthermore, CEA, ICAM-1 and CD44v6 were increased by TNF-alpha plus calcitriol
Myers et al., Am J Physiol 1992 : We conclude from these data that although PMA induced ICAM-1 expression may be triggered through activation of protein kinase C, ICAM-1 induction by IL-1 beta, TNF-alpha , or LPS may involve distinct regulatory pathway ( s )
Möst et al., J Immunol 1992 : In contrast, ICAM-1 expression on two of the three cell lines ( KG1 and HL60 ) was inducible by TNF-alpha to a much higher degree than by IFN-gamma, whereas the results with U937 were comparable with those obtained for normal monocytes
Mattila et al., Scand J Immunol 1992 : TNF alpha induced expression of endothelial adhesion molecules, ICAM-1 and VCAM-1, is linked to protein kinase C activation
Sabatini et al., Immunol Lett 2003 : Fluticasone propionate, at the two concentrations tested ( 10 and 100 nM ), was more effective in inhibiting the ` IL-4 plus TNF-alpha induced ' ICAM-1 expression than VCAM-1 expression ( P < 0.05 ) and in downregulating RANTES than IL-8 or GM-CSF secretion ( P < 0.05 )
Dvorin et al., J Heart Valve Dis 2003 : HPVEC express E-selectin, ICAM-1 , and VCAM-1 in response to the inflammatory cytokine tumor necrosis factor-alpha (TNF-alpha) similarly to other types of cultured human endothelial cells
VandenBerg et al., J Cell Biochem 2004 : TNF-alpha increased VCAM-1, ICAM-1 , and E-selectin by 4 h but had no affect on the expression of PECAM
Choi et al., J Nutr 2004 : TNF-alpha significantly induced HUVEC protein expression of VCAM-1, ICAM-1 , and E-selectin with increasing mRNA levels
Huang et al., Carcinogenesis 2004 (Neoplasm Invasiveness) : In Matrigel assays, ICAM-1 and COX-2 expressions induced by TNF-alpha elicited A549 and NCI-H292 cell invasion, respectively, and these effects were inhibited by both compounds
Jiang et al., Life Sci 2004 : The mRNA expression of E-selectin, ICAM-1 and VCAM-1, and activation of NF-kappaB induced by TNF-alpha for 2 h were significantly decreased by the above two pretreatments
Costa et al., Xenotransplantation 2004 : We first evaluated by flow cytometry the pTNFR1Ig capacity to prevent TNF alpha induced expression of SLAI, SLAII, VCAM-1, ICAM-1 and E-selectin on the cell surface of porcine aortic endothelial cells ( PAEC )
Zhang et al., J Am Soc Nephrol 2005 : This hypothesis was examined further by stimulation of PTC ICAM expression by TNF-alpha ... Although stimulation of PTC by BMP-7 alone decreased cell surface ICAM expression, it did not affect TNF-alpha induced ICAM expression
Harada et al., Nephron. Experimental nephrology 2005 : In these cells, ICAM-1 and E-selectin expression was up-regulated by TNF-alpha , as in native cultured HGEC
Stangl et al., J Nutr 2005 : Phloretin prevented TNF-alpha stimulated upregulation of VCAM-1, ICAM-1 , and E-selectin expression in a concentration dependent manner
Sasaki et al., BMC physiology 2005 : Trogliazone ( 20 microM ) reduced TNF-alpha induced VCAM-1, ICAM-1 and E-selectin expression
Jiang et al., Respir Med 2005 : The mRNA expression of E-selectin, ICAM-1 and VCAM-1, and activation of NF-kappaB induced by TNF-alpha for 2 h were decreased significantly by the above two pretreatments
Ogawara et al., Arthritis Res Ther 2006 : RGD targeted adenovirus delivered the dnIkappaB via alphavbeta3 to become functionally expressed, leading to complete abolishment of TNF-alpha induced up-regulation of E-selectin, ICAM-1 , VCAM-1, IL-6, IL-8, VEGF-A and Tie-2
Mo et al., J Ethnopharmacol 2007 : Evidence presented in this report demonstrated that CML-1 inhibited the adhesive capacity of HUVEC and the TNF-alpha mediated induction of E-selectin, ICAM-1 and VCAM-1 in HUVEC by inhibiting the IkB/NF-kB signaling pathway at the level of IkB kinase, which may explain the ability of CML-1 to suppress inflammation and modulate the immune response
Conover et al., Growth Horm IGF Res 2008 : TNF-alpha induced VCAM, ICAM , and MCP-1 expression ( 4h ) preceded PAPP-A expression ( 24h )
Seldon et al., J Immunol 2007 (Inflammation) : Basal and TNF induced expression of VCAM-1, ICAM-1 , and E-selectin were increased in Hmox1 ( -/- ) vs Hmox1 ( +/+ ) EC, an effect reversed by Fe chelation using deferoxamine mesylate ( DFO )
Hung et al., Eur J Pharmacol 2008 : Taken together, we provided here the first evidence showing that lycopene is able to inhibit TNF-alpha induced NF-kappaB activation, ICAM-1 expression, and monocyte-endothelial interaction, suggesting an anti-inflammatory role of lycopene and possibly explaining in part why lycopene can prevent cardiovascular diseases
Winterbone et al., Atherosclerosis 2009 : Quercetin was able to reduce TNFalpha induced upregulation of VCAM-1, ICAM-1 and MCP-1 at both the protein and transcript ( mRNA ) level in HUASMC
Wang et al., J Perinatol 2009 (Pre-Eclampsia) : ( 1 ) TNFalpha induced dose dependent increases in ICAM, VCAM and E-selectin expressions in endothelial cells ; ( 2 ) Digibind could attenuate and reduce TNFalpha induced upregulation of endothelial E-selectin, ICAM and VCAM expressions
Ming et al., BMC cardiovascular disorders 2009 : The induction of the expression of endothelial VCAM-1, ICAM-1 and E-selectin by TNFalpha was concentration-dependently reduced by incubation of the endothelial cells with the arginase inhibitor L-norvaline
Yu et al., J Agric Food Chem 2010 : Induction of heme oxygenase 1 and inhibition of tumor necrosis factor alpha induced intercellular adhesion molecule expression by andrographolide in EA.hy926 cells
Chen et al., Vascul Pharmacol 2011 : ZLJ-6, a novel COX/5-LOX inhibitor, attenuates TNF-a induced endothelial E-selectin, ICAM-1 and VCAM-1 expression and monocyte-endothelial interactions via a COX/5-LOX independent mechanism
Escobar et al., Biochem Pharmacol 2012 (Pancreatitis, Acute Necrotizing) : Oxidative stress significantly contributes to the TNF-a induced up-regulation of Icam and iNos in AR42J cells
Xing et al., Pharm Biol 2013 : In addition, the effects of total flavonoids on inflammatory molecule expression of cells were tested by immunohistochemistry staining, showing that TNF-a induced expression of PCNA, NF-?B p65, ICAM-1 , and VCAM-1 in VSMCs was dose-dependently suppressed by total flavonoids
Kong et al., Lipids in health and disease 2013 : However, comparing to the model rabbits, levels of TNF-a , IL-1ß and MDA decreased significantly and serum SOD activity increased , gene and protein expressions of E-sel, ICAM-1 , VCAM-1 and MCP-1 in aortas decreased significantly with the treatment of kaempferol
Tang et al., Am J Physiol 1995 : In conclusion, 1 ) CD11a, ICAM-1 , and CINC play major roles in the LPS initiated emigration of PMNs into the bronchoalveolar space, and 2 ) the TNF that drives ICAM-1 and CINC expression is derived largely from alveolar macrophages rather than PMNs
Ikeda et al., J Cardiovasc Pharmacol 1994 : In ELISA analysis, ICAM-1 molecule expression on myocytes was significantly stimulated by TNF-alpha ( 100 U/ml ), but not by IL-6 ( 100 U/ml ) or IL-8 ( 100 ng/ml ) dose dependently
Groves et al., Br J Dermatol 1995 (Dermatitis...) : TNF-alpha induced endothelial E-selectin, intercellular adhesion molecule-1 ( ICAM-1 ) and vascular cell adhesion molecule-1 ( VCAM-1 ) in all groups, and adhesion molecule expression by interstitial dermal dendritic cells ( ICAM-1 and VCAM-1 ) and keratinocytes ( ICAM-1 ) was observed
Ferran et al., Biochem Biophys Res Commun 1995 : We show that pyrrolidine dithiocarbamate strongly reduces the TNF alpha mediated induction of E-selectin, VCAM-1, ICAM-1 , PAI-1, tissue factor, IL-8 and I kappa B-alpha
Goebeler et al., Immunobiology 1993 (Inflammation) : Furthermore, ICAM-1 expression by bone marrow derived M phi could be up-regulated by tumor necrosis factor-alpha , interferon-gamma and LPS
Ford et al., J Surg Res 1996 : Labeling had no effect on EC proliferation rates nor on TNF induced upregulation of ICAM expression
Syrbu et al., Exp Lung Res 1996 (Disease Models, Animal...) : The time courses for ICAM and ELAM stimulation with pure TNF alpha or BALF60 containing equivalent levels of TNF alpha were the same
Klein et al., Pathobiology 1995 : Thus, ICAM-1 increased dramatically after 5 h incubation with TNF-alpha
Gille et al., J Clin Invest 1997 (Inflammation) : To determine modes of retinoid action in the modulation of inflammatory responses, we explored effects of all-trans-retinoic acid ( t-RA ) on the TNFalpha induced expression of vascular cell adhesion molecule-1 ( VCAM-1 ), intercellular adhesion molecule-1 ( ICAM-1 ), and E-selectin in cultured human dermal microvascular endothelial cells
Fujisawa et al., J Dermatol Sci 1997 (Carcinoma, Squamous Cell) : This evidence contrasts with previously reported findings indicating that, in human cultured keratinocytes, ICAM-1 expression is induced by IFN-gamma and TNF-alpha , and not by either IL-1 or LPS
Jiang et al., Neurochem Int 1997 : Since adhesion molecules ( AM ) induced on endothelial cells ( EC ) play an important role in T-cell migration into the CNS, the objective of this study was to examine the effect of IFN beta-1b on the expression of the AM, ICAM-1 , V-CAM and E-selection induced on EC by IFN-gamma, TNF-alpha , or IL-1 beta
Shen et al., J Virol 1997 : TNF-alpha significantly enhanced WNV induced increases in E-selectin, P-selectin, ICAM-1 , and VCAM-1 expression, while IFN-gamma enhanced WNV induced ICAM-1 expression
Braun et al., Arterioscler Thromb Vasc Biol 1997 (Second Messenger Systems) : In cultured human smooth muscle cells from coronary arteries and saphenous veins, tumor necrosis factor-alpha (TNF-alpha) and interleukin-1 beta (IL-1 beta) induced the expression of intercellular adhesion molecule ( ICAM-1 ) and vascular cell adhesion molecule ( VCAM-1 ), whereas interferon-gamma (INF-gamma) selectively stimulated the expression of ICAM-1
Takeshita et al., Infect Immun 1998 : In addition, ICAM-1 clearly inhibited the fimbria induced expression of IL-1beta and TNF-alpha genes in the cells
Chatterjee et al., Arterioscler Thromb Vasc Biol 1998 (Arteriosclerosis) : Additional studies have revealed that LacCer mediates the tumor necrosis factor-alpha (TNF-alpha) induced nuclear factor-kappaB expression and intercellular adhesion molecule ( ICAM-1 ) expression in vascular endothelial cells via the redox dependent transcriptional pathway
Lakshminarayanan et al., J Biol Chem 1998 : We previously demonstrated that tumor necrosis factor-alpha (TNFalpha) and H2O2 differentially regulate interleukin-8 (IL-8) and intercellular adhesion molecule ( ICAM-1 ) gene expression in endothelial and epithelial cells