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JUN — TRNAE1

Text-mined interactions from Literome

Rourke et al., FEBS Lett 1999 : As previously noted, c-Jun/c-Fos heterodimers greatly increase promoter activity through association with the CRE/TRE
Cho et al., Infect Immun 2002 (Scrub Typhus...) : Taken together, AP-1 activation by both the ERK pathway and the p38 MAPK pathway as well as their binding to TRE at position -69 to -63 in proximal promoter and TRE at position -2276 to -2270 in enhancer region is altogether essential in induction of MCP-1 mRNA in HUVEC infected with O. tsutsugamushi
Nagashima et al., Mol Cell Endocrinol 2003 (Pituitary Neoplasms) : Accordingly, PACAP stimulated AP-1 and CREB transcriptional activity and E ( 2 ) inhibited TRE-LUC but not CRE-LUC activation
de Groot et al., Oncogene 1992 : In transient transfection experiments, TRE activation by Jun is strongly enhanced by co-transfection of the catalytic subunit of PKA or forskolin treatment, although not in all cell types studied
de Groot et al., Mol Cell Biol 1991 (Cell Transformation, Viral) : AP-1 binds to 12-O-tetradecanoylphorbol-13-acetate ( TPA ) -responsive elements ( TREs ) ; therefore, E1A might modulate a specific signal transduction pathway normally induced by activation of the protein kinase C. Binding of jun/AP-1 to a TRE is induced in all cell types studied when E1A is expressed