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CCL3 — IL15

Text-mined interactions from Literome

Wang et al., Clin Exp Immunol 2003 (Arthritis, Rheumatoid) : MIP-1alpha production by synovial CD4+ T cells was also enhanced by IL-15 ... Taken together, these data suggest CCR5+CD4+ T cells infiltrate the inflamed synovium and IL-15 up-regulates CCR5 and CD40L expression further and enhance MIP-1alpha production in synovial CD4+ T cells
Waehre et al., J Am Coll Cardiol 2003 (Coronary Artery Disease) : Our main findings were : 1 ) gene expression of several chemokines ( i.e., macrophage inflammatory protein [MIP ] -1alpha, MIP-1beta, and interleukin [ IL]-8 ) and chemokine receptors ( i.e., CC chemokine receptor [ CCR]1, CCR2, CCR4, and CCR5 ) was markedly increased among CAD patients compared with healthy control subjects ; 2 ) treatment with atorvastatin and simvastatin markedly reduced the mRNA levels of some of these chemokines ( i.e., MIP-1alpha, MIP-1beta, IL-8 ) and receptors ( i.e., CCR1 and CCR2 ), with the most pronounced effect in the atorvastatin group ; and 3 ) statin therapy reduced the spontaneous release of IL-8 and MIP-1alpha from PBMCs in CAD patients
Forcina et al., Cytokine 2004 (HIV Infections) : Finally, IL-15 had positive effects on RANTES, MIP-1alpha , and MIP-1beta release by CD4 ( + ) and CD8 ( + ) T cells
Verri et al., Eur J Immunol 2007 (Arthritis, Rheumatoid) : Consistent with this observation, IL-15 induced IL-18 production, and IL-15 or IL-18 injection induced the production of MIP-2, MIP-1alpha , TNF-alpha and LTB4
Wang et al., Arthritis Rheum 2013 (Intervertebral Disc Degeneration...) : Tumor necrosis factor a- and interleukin-1ß dependent induction of CCL3 expression by nucleus pulposus cells promotes macrophage migration through CCR1
Bluman et al., J Clin Invest 1996 : Fresh NK cells produce negligible amounts of MIP-1 alpha after stimulation with the monocyte derived cytokines IL-12, TNF-alpha, IL-1 beta, or IL-10, while stimulation with IL-15 alone results in modest MIP-1 alpha production