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NGF — PDCD6
Text-mined interactions from Literome
Frade et al., Microsc Res Tech 1999
:
Thus, NT-3 promotes the conversion of neuroepithelial cells into neurons, whereas BDNF and
NGF control the
programmed cell death ( apoptosis ) that affects early postmitotic neuroblasts
Mayumi-Matsuda et al., Biochim Biophys Acta 1999
:
Depletion of
nerve growth factor (NGF) from differentiated, neuronal PC12 cells
causes a form of
programmed cell death that stems from the attenuation of NGF receptor signaling and the resultant expression of certain genes required for cell death
Gentry et al., J Biol Chem 2000
(Neurilemmoma) :
In contrast to its trophic actions through TrkA,
NGF binding to p75 has been shown to
activate programmed cell death through a mechanism involving the stress kinase JNK
Deshmukh et al., J Cell Biol 2000
:
Nerve growth factor (NGF) deprivation
induces a Bax dependent, caspase dependent
programmed cell death in sympathetic neurons
Niederhauser et al., J Neurosci Res 2000
:
In SK-N-MC cells this leads to inhibition of
NGF induced
programmed cell death
Peltier et al., Curr Opin Neurol 2002
(Peripheral Nervous System Diseases) :
In contrast,
nerve growth factor prevents initiation of the
programmed cell death associated with cisplatin neurotoxicity
Jelaso et al., Int J Dev Neurosci 2005
:
NGF , a neurotrophic factor best known for its role in promoting cell survival,
regulates many neurodevelopmental processes, including synaptic plasticity, neurite outgrowth and
programmed cell death
Ito et al., J Neurochem 1995
:
Ceramide prevents neuronal
programmed cell death induced by
nerve growth factor deprivation
Mills et al., J Neurochem 1996
:
Terminally differentiated PC12 cells are a useful neuron-like model for studying
programmed cell death in
response to
nerve growth factor (NGF) deprivation
Frade et al., Development 1999
:
The
role of
nerve growth factor (NGF) and of the neurotrophin receptor p75 ( p75 ( NTR ) ) in
programmed cell death was investigated in the retina and the spinal cord of mouse embryos
Sanz et al., Oncogene 1999
:
The increase in c-Raf levels also prevented
nerve growth factor (NGF) dependent induction of
programmed cell death