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GRIA1 — PRKAR2B

Text-mined interactions from Literome

Vinade et al., Cell Mol Neurobiol 2000 : Prior incubation of PSDs under dephosphorylating conditions results in only a small PKA mediated phosphorylation of GluR1
Mangiavacchi et al., J Neurochem 2004 : Post-natal rat nucleus accumbens (NAc) cultures were used to study the role of protein kinase A (PKA) in D1 receptor regulation of the surface expression of the AMPA receptor subunit GluR1
Harris et al., Neurosci Lett 2004 : Immunoblot analysis of perirhinal cortical neurons using GluR1 and GluR1-S845 phosphorylation state specific antibodies showed that stimulation of adenylyl cyclase ( AC ) with forskolin ( FSK ) dramatically increased PKA mediated phosphorylation of GluR1-S845
Vanhoose et al., J Neurosci 2006 : After investigating the effect of coactivation of the NMDAR and the Gs-coupled beta-adrenergic receptor on GluR1 phosphorylation state, we have observed a novel signal that prevents PKA mediated phosphorylation of GluR1 at serine site 845
Lu et al., EMBO J 2007 : Other studies demonstrate that GluR1 homomeric AMPA receptors are the main GluR2 lacking AMPA receptors in adult hippocampus and require PKA for their functional postsynaptic expression during potentiation
Ahn et al., J Pharmacol Exp Ther 2009 : Interactions of protein kinases activated by intracellular Ca ( 2+ ) release downstream to PLC modulate the phosphorylation state of GluR1 on Ser831 and Ser845 : phosphorylation of GluR1 on Ser831 is up-regulated by the protein kinase C and calcium-calmodulin dependent protein kinase ( CaMK ) /c-Jun N-terminal kinase (JNK) pathways, whereas phosphorylation of GluR1 on Ser845 is up-regulated by the protein kinase A (PKA), PKA/ERK1/2 , and PKA/JNK pathways
Wang et al., FASEB J 2010 : The binding is required to induce G-protein/cAMP/protein kinase A (PKA) signaling, which controls PKA dependent phosphorylation of GluR1 and beta ( 2 ) AR, and AMPA receptor mediated excitatory postsynaptic currents ( EPSCs )
Cui et al., Learn Mem 2011 : GluA1 PKA dependent phosphorylation peaked 10 min after the 10-min training trial and returned to baseline within 90 min ... PKA mediated GluA1 phosphorylation and later GluA1 insertion could, conjointly, provide increased AMPA function to support both short-term and long-term appetitive memory
Dell'anno et al., Neuropharmacology 2013 : The ability of DHPG to increase PKA dependent phosphorylation of GluA1 depends on concomitant activation of the dopamine- and cAMP regulated phosphoprotein of 32kDa ( DARPP-32 )