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E2F1 — NCOA3

Pathways - manually collected, often from reviews:

• FastForward regulation: E2F1 → NCOA3 (transcriptional regulation, increase) Mussi et al., Mol Endocrinol 2006*
  Evidence: REG, PROMACTIVITY, DNABINDING

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Bind Interaction: NCOA3 — E2F1 Louie et al., Mol Cell Biol 2004*
• IRef Bind_translation Interaction: NCOA3 — E2F1 (coimmunoprecipitation) Louie et al., Mol Cell Biol 2004*
• IRef Bind_translation Interaction: NCOA3 — E2F1 (affinity chromatography technology) Louie et al., Mol Cell Biol 2004*
• IRef Biogrid Interaction: NCOA3 — E2F1 (colocalization, imaging technique) Louie et al., Mol Cell Biol 2006*
• IRef Biogrid Interaction: NCOA3 — E2F1 (physical association, affinity chromatography technology) Louie et al., Mol Cell Biol 2004*
• IRef Biogrid Interaction: NCOA3 — E2F1 (direct interaction, pull down) Louie et al., Mol Cell Biol 2004*
• IRef Hprd Interaction: NCOA3 — E2F1 (in vivo) Louie et al., Mol Cell Biol 2004*
• IRef Hprd Interaction: NCOA3 — E2F1 (in vitro) Louie et al., Mol Cell Biol 2004*
• IRef Ophid Interaction: NCOA3 — E2F1 (aggregation, interologs mapping) Brown et al., Bioinformatics 2005

Text-mined interactions from Literome

Louie et al., Mol Cell Biol 2004 (Breast Neoplasms) : We also demonstrated, by adenovirus vector mediated RNA interference, that ACTR is required for E2F1 mediated gene expression and the proliferation of estrogen receptor ( ER ) -negative breast cancer cells
Mussi et al., Mol Endocrinol 2006 : Furthermore, overexpression of E2F1 significantly increases the promoter activity of the endogenous SRC-3 gene and boosts SRC-3 expression in vivo