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EGFR — TMED7

Text-mined interactions from Literome

Kiyota et al., Oncology 2002 (Carcinoma, Squamous Cell...) : These results suggested that the antiproliferative effect of EGFR blockade by mAb225 in oral SCC may be mediated by p27 ( KIP1 ) and p15(INK4B)
Lee et al., American journal of physiology. Renal physiology 2008 : Consequently, the inhibition of Ca ( 2+ ), PKC, EGFR , p44/42 MAPKs, or NF-kappaB blocked the BSA induced increases in cyclin D1, cyclin dependent kinase (CDK)4, cyclin E, or CDK2 and restored the BSA induced inhibition of p21 ( WAF/Cip1 ) and p27 ( Kip1 ) expression
Migita et al., Int J Oncol 2008 (Carcinoma, Renal Cell...) : These results suggest that E-cadherin mediated adhesion may be involved in the contact stimulation for cell proliferation in part through the downregulation of p27 and the activation of EGFR in human cancers
White et al., Dev Biol 2012 : In addition, we show that EGFR signaling in p75+ mouse supporting cells is required for the down-regulation of the cell cycle inhibitor p27 ( Kip1 ) ( CDKN1b ) to enable cell cycle re-entry
Jameson et al., J Oral Pathol Med 2013 (Carcinoma, Squamous Cell...) : Activation of the insulin-like growth factor-1 receptor alters p27 regulation by the epidermal growth factor receptor in oral squamous carcinoma cells
Peng et al., Cancer Res 1996 (Prostatic Neoplasms) : These studies demonstrate that the antiproliferative effect of EGFR blockade in DU145 cells may be mediated by up-regulation of p27KIP1 at both the mRNA and protein levels