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ATP5O — CSF2

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Borsody et al., Headache 2006 (Pseudotumor Cerebri) : Since the production of CSF is dependent upon sodium-potassium ATPase activity, and since there is evidence that the increased intracranial pressure found in the condition of IIH may be the result of increased CSF production, we hypothesized that the level of endogenous OLC would be reduced in the CSF of patients with IIH
Han et al., BMC neuroscience 2009 : Moreover, we also showed that adenosine receptor signaling can regulate the production of CSF by controlling the expression of Na ( + ), K ( + ) -ATPase and CBF
Halperin et al., J Biol Chem 1988 : Human cerebrospinal fluid (CSF) inhibits the Na+/K+ pump in human red cells and the activity of purified Na+/K+-ATPase ( Halperin, J. A., Shaeffer, R., Galvez, L., and Malavé, S. ( 1985 ) Proc. Natl. Acad. Sci. U.S. A. 80, 6102-6104, 1983 ; Halperin, J. A., Martin, A. M., and Malavé, S. ( 1985 ) Life Sci. 37, 561-566
Niessen et al., Blood 1997 : Our findings suggested two coordinate mechanisms by which the activity of the v-ATPase could be increased by G-CSF : the synthesis of cytosolic v-ATPase subunits and their translocation to the membrane