Gene interactions and pathways from curated databases and text-mining

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CAMK2D — CREB1

Pathways - manually collected, often from reviews:

  • OpenBEL Selventa BEL large corpus: CREB1 → CAMK2D (directlyIncreases, CAMK2D Activity, CREB1 Activity)
    Evidence: The substrate preferences of CaMKI and CaMKIV are similar and intersect with CaMKII. In vitro, all three can phosphorylate synapsin I, cAMP response element-binding protein (CREB), and activating transcription factor 1 (11, 12).
  • BioCarta transcription factor creb and its extracellular signals: CAMKII/CaM/Ca ++/CaM/Ca ++/CaM/Ca ++/CaM/Ca ++ complex (CAMK2A_CAMK2G_CAMK2B_CAMK2D-CAMK2B) → CREB/CREB complex (CREB1) (modification, activates)
  • BioCarta ca-calmodulin-dependent protein kinase activation: CAMKII/CaM/Ca ++/CaM/Ca ++/CaM/Ca ++/CaM/Ca ++ complex (CAMK2A_CAMK2G_CAMK2B_CAMK2D-CAMK2B) → CREB (CREB1) (modification, activates)
  • Reactome Reaction: CREB1 → CAMK2D (reaction) Wu et al., J Biol Chem 2001*