◀ Back to FGF22
FGF22 — FGF8
Pathways - manually collected, often from reviews:
-
Reactome Reaction:
FGF8
→
FGF22
(reaction)
Carpenter et al., Exp Cell Res 1999, Wong et al., Proc Natl Acad Sci U S A 2002, Lax et al., Mol Cell 2002, Fong et al., J Biol Chem 2003, Agazie et al., Oncogene 2003, Takeda et al., Clin Cancer Res 2007, Ahmed et al., Biochem J 2008, Schüller et al., Biochem J 2008, Qing et al., J Clin Invest 2009, Turner et al., Nat Rev Cancer 2010, Bai et al., Cancer Res 2010, Dutt et al., PloS one 2011, Wesche et al., Biochem J 2011, Klint et al., J Biol Chem 1995, Wang et al., Mol Cell Biol 1994, Ong et al., Biochem Biophys Res Commun 1996, Kanai et al., J Biol Chem 1997, Kouhara et al., Cell 1997, Ong et al., Biochem Biophys Res Commun 1997, Hadari et al., Mol Cell Biol 1998, Raffioni et al., J Biol Chem 1998
Text-mined interactions from Literome
Liu et al., Development 2002
(Limb Deformities, Congenital) :
In this system,
FGF10 but not FGF8 protein injected into the mutant distal tip mesenchyme
restores Fgf8 expression in the AER
Saitsu et al., Mech Dev 2006
(Heart Defects, Congenital) :
Moreover, over-expression of
Fgf15 resulted in up-regulation of
Fgf8 expression in the isthmus/r1
Katayama et al., Int J Oncol 2010
(Prostatic Neoplasms) :
Overall, bicalutamide inhibits the cyclin A expression possibly by inhibiting
FGF-8 mRNA expression and FGF-8 protein secretion but not by
inhibiting FGF receptor ( FGFR ) signalling in androgen dependent cell lines, and by other mechanisms in androgen independent cell lines
Miyake et al., Biology open 2013
:
fgf3 and
fgf8 were expressed earlier than fgf22 in the MHB primordium and Fgf3/Fgf8 signaling was
required for
fgf22 expression in the posterior midbrain