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GFAP — LIF

Text-mined interactions from Literome

Wright et al., J Neurochem 2003 : Protein analysis confirmed LIF regulated expression of glial fibrillary acidic protein , CD44, and major histocompatibility complex I
Shen et al., Mol Cell Neurosci 2004 : However, leukemia inhibitory factor (LIF) could stimulate GFAP expression suggesting that astrocytes not derived from radial glia are less affected by absence of GAP-43
Pitman et al., Mol Cell Neurosci 2004 : Exogenous LIF abrogated neurosphere formation and promoted expression of GFAP by all cells in wild-type and heterozygous cultures
Emery et al., Mol Cell Neurosci 2006 : Proliferating NPC cultures can be generated from SOCS3 knockout ( SOCS3KO/KO ) embryos and display prolonged STAT3 phosphorylation and induction of the GFAP gene in response to LIF
Mothe et al., PloS one 2011 : Leukemia inhibitory factor (LIF) promotes NSPC proliferation and significantly enhances GFAP expression in hypoxia
Kahn et al., Glia 1994 : Using Northern and Western analyses in conjunction with immunocytochemistry we have demonstrated that ciliary neurotrophic factor (CNTF), leukemia inhibitory factor (LIF) , and interleukin-6 (IL-6) cause a transient increase in glial fibrillary acidic protein (GFAP) in oligodendrocyte type-2 astrocyte ( O-2A ) progenitor cells
Nishiyama et al., Neurosci Lett 1993 : Regulation of glial fibrillary acidic protein in serum-free mouse embryo ( SFME ) cells by leukemia inhibitory factor and related peptides
Yoshida et al., Brain Res Dev Brain Res 1993 : Leukemia inhibitory factor (LIF) and ciliary neurotrophic factor (CNTF) induced glial fibrillary acidic protein (GFAP) and decreased A2B5 antigen in AP-16 cells, indicating that these cytokines induced AP-16 cells to differentiate into astrocytes