◀ Back to ICAM1

CD8A — ICAM1

Text-mined interactions from Literome

Dohlsten et al., Eur J Immunol 1991 : CD3+CD4+ and CD3+CD8+ cytotoxic T cell lines were equally dependent on the expression of ICAM-1 on the target cell
Papasavvas et al., AIDS 2008 (Chronic Disease...) : We assessed changes in plasma levels of von Willebrand factor, soluble vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 by enzyme linked immunosorbent assay, as well as T-cell activation ( CD8+/CD38+, CD8+/HLA-DR+ and CD3+/CD95+ ) by flow cytometry, in 36 chronically HIV-1 infected patients participating in a randomized study ... We assessed changes in plasma levels of von Willebrand factor, soluble vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 by enzyme linked immunosorbent assay, as well as T-cell activation ( CD8+/CD38+ , CD8+/HLA-DR+ and CD3+/CD95+ ) by flow cytometry, in 36 chronically HIV-1 infected patients participating in a randomized study
Viaud et al., J Immunother 2011 : Here, we show that interferon-? is a key cytokine conditioning the dendritic cell to induce the expression of CD40, CD80, CD86, and CD54 on Dex, endowing them with direct and potent peptide dependent CD8 ( + ) T-cell triggering potential in vitro and in vivo
Fauconnier et al., Am J Pathol 2011 (Brain Ischemia...) : Recruitment and activation of CD8 ( + ) T cells, and ICAM-1 and CD69 expression were reduced in the brain of resistant mice ; however, the pulmonary inflammation and edema associated with PbA infection were still present in the absence of functional PKC-?
Deeths et al., Eur J Immunol 1999 : The CD8+ T cell response to ICAM-1 costimulation is associated with increased proliferation and IL-2 production at levels similar to those seen with B7-1 costimulation, but clonal expansion in response to ICAM-1 is not as great due to decreased cell survival