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IL1R1 — TLR7

Text-mined interactions from Literome

Cheung et al., J Immunol 2005 : However, AcPL is dispensable for the activation or inhibition of IL-1R and the various TLR signals that we have examined
Zhang et al., J Immunol 2009 (Multiple Sclerosis, Relapsing-Remitting) : We found that IFN-beta1a in vitro treatment of human monocyte derived DCs induced the expression of TLR7 and the members of its downstream signaling pathway, including MyD88, IL-1R associated kinase 4, and TNF receptor associated factor 6, while it inhibited the expression of IL-1R ... Using small interfering RNA TLR7 gene silencing, we confirmed that IFN-beta1a induced changes in MyD88, IL-1R associated kinase 4, and IL-1R expression were dependent on TLR7