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CCL3 — IL1A
Text-mined interactions from Literome
Netea et al., Eur J Immunol 2000
(Chlamydia Infections) :
In contrast, synthesis of MCP-1 and
MIP-1alpha was
dependent on endogenous TNF, but not
IL-1
Guo et al., J Neurochem 2003
:
We report herein that human neuronal cells ( NT2-N ) produce
macrophage inflammatory protein-1alpha and -1beta ( MIP-1alpha and MIP-1beta ), which could be
enhanced by
interleukin (IL)-1beta at both mRNA and protein levels
Lu et al., Am J Pathol 2003
(Carcinoma, Hepatocellular...) :
Four hepatoma cell lines produced
CCL3 only in
response to
interleukin (IL)-1 alpha and IL-1 beta ... Thus,
IL-1 may
enhance the local production of
CCL3 , which may interact with CCR1 expressed on hepatoma cells, in an autocrine and/or paracrine manner
Nakamura et al., Clin Exp Rheumatol 2007
(Arthritis, Rheumatoid...) :
Celecoxib increased
interleukin-1 (IL-1) induced production of RANTES and
MIP-1alpha by chondrocytes and decreased IL-1 induced NO production by chondrocytes, whereas it did not affect MMP production
Zhang et al., J Biol Chem 2010
:
CCAAT/enhancer binding protein ß and NF-?B mediate high level expression of chemokine genes
CCL3 and CCL4 by human chondrocytes in
response to
IL-1ß
Alexander et al., Physiol Genomics 2012
(Inflammation...) :
Finally, immunofluorescent staining of mouse atherosclerotic lesions revealed the presence of cells positive for the marker of an
IL-1ß stimulated inflammatory SMC,
chemokine (C-C motif) ligand 20 ( CCL20 ), but not the PDGF-DD induced gene, regulator of G protein signaling 17 ( RGS17 )
Kitahara et al., Inflamm Res 2012
(Arthritis, Rheumatoid) :
The mRNA expressions of
CCL3 , CCL4, and CXCL8 were
up-regulated by
IL-1ß and down-regulated by tacrolimus
Hosaka et al., Clin Exp Immunol 1994
(Arthritis, Rheumatoid) :
Cultured synovial fibroblasts were found capable of expressing and producing IL-8, GRO, monocyte chemotactic and activating factor ( MCAF ),
macrophage inflammatory protein-1 alpha ( MIP-1 alpha ), MIP-1 beta and RANTES in
response to
IL-1 alpha