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RHOB — TGFB3

Text-mined interactions from Literome

Moustakas et al., J Cell Sci 1999 (Cell Transformation, Neoplastic) : Furthermore, transforming growth factor beta-1 upregulates considerably the levels of the RhoB small GTPase and less the RhoA levels
Nishikimi et al., J Hypertens 2004 (Glomerulosclerosis, Focal Segmental...) : After 7 weeks, untreated DS were characterized by decreased kidney function, increased proteinuria, abnormal morphological findings, increased adrenomedullin and atrial natriuretic peptide (ANP) levels, and increased renal messenger RNA expression of RhoB , Rho-kinasealpha, Rho-kinasebeta, collagen I and collagen III, and transforming growth factor-beta ( TGF-beta ) in the renal cortex compared with DR. Chronic fasudil treatment significantly improved renal function ( serum creatinine, -26 % ; blood urea nitrogen, -41 % ; creatinine clearance, +42 % ), proteinuria ( -24 % ) and histological findings ( glomerular injury score, -49 % ; afferent arteriolar injury score, -17 % ) without changing blood pressure compared with untreated DS
Engel et al., J Biol Chem 1998 : RhoB is stabilized by transforming growth factor beta and antagonizes transcriptional activation