◀ Back to CA2

CA2 — VWF

Text-mined interactions from Literome

Yoshioka et al., Ann N Y Acad Sci 2001 : We have established an assay system using streptolysin-O permeabilized platelets to analyze the Ca2+ induced secretions of von Willebrand factor stored in alpha-granules and [ 3H ] 5-hydroxytryptamine ( 5-HT ) in dense-core granules
Carew et al., Biochem J 1992 : The roles of protein kinase C and intracellular Ca2+ in the secretion of von Willebrand factor from human vascular endothelial cells
Cleator et al., Blood 2006 (Calcium Signaling) : VWF release can be stimulated by both Ca2+ and cAMP, and, although both VWF and P-selectin are found in Weibel-Palade bodies ( WPBs ), we found that their release could be differentially regulated
Francesconi et al., Biochem Biophys Res Commun 1995 : Type IIB von Willebrand factor induces phospholipase A2 activation and cytosolic Ca2+ increase in platelets
Mazzucato et al., Thromb Haemost 1996 (Second Messenger Systems) : The inhibition of P-vWF binding to GP Ib, obtained with anti GP Ib monoclonal antibody ( LJ-Ib1 ), inhibits the increase of intracellular Ca2+ concentration ([Ca2+]i) and platelet aggregation
Iouzalen et al., Cell Calcium 1996 : In conclusion, we demonstrate that, in the absence of Ca2+ entry from the extracellular space, the [Ca2+ ] i increase elicited by SK & F 96365 or thapsigargin is not sufficient to initiate NO synthesis and vWF secretion
van den Eijnden-Schrauwen et al., Arterioscler Thromb Vasc Biol 1997 : In this study, we investigated the role of Ca2+ and G proteins in thrombin induced acute release ( regulated secretion ) of tissue-type plasminogen activator ( TPA ) and von Willebrand factor (vWF) , using a previously described system of primary human umbilical vein endothelial cells ( HUVECs )
Milner et al., J Lab Clin Med 1998 : Binding of vWf to platelet Gplb was responsible for the ( Ca2+ ) i signal ... The present findings are a more accurate representation of the vWf induced platelet ( Ca2+ ] i signal
Nilius et al., Verhandelingen - Koninklijke Academie voor Geneeskunde van België 1998 (Second Messenger Systems) : Ca2+ signals in endothelial cells play the key role in the release of NO, prostacyclin ( PGI2 ), platelet activating factor (PAF), von Willebrand factor (vWF), tissue plasminogen activator (tPA) and tissue factor pathway inhibitor (TFPI)