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ESR2 — FOS
Text-mined interactions from Literome
Planas-Silva et al., Cancer Res 1999
(Breast Neoplasms) :
Other transcriptional cofactors that allow
estrogen receptor to
induce expression of
AP-1 may be required for estrogen to act as a mitogen
Wu et al., Eur J Endocrinol 2000
:
The results suggest that, during gestation, myometrial
ERbeta may
inhibit AP-1 activity and thus block induction of the cx43 gene and other labor associated genes
Petz et al., Endocrinology 2002
(Breast Neoplasms) :
Purified
estrogen receptor ( ER ) enhanced binding of
Fos and Jun to the +90 AP-1 site and bound to an adjacent imperfect ERE half-site
Kanda et al., J Invest Dermatol 2003
:
Transfection of
ERbeta enhanced the inhibitory effects of E2 on Sp1 and
AP-1 transcriptional activities and MCP-1 promoter activities in nontransfected keratinocytes ... These results suggest that E2-bound
ERbeta may inhibit MCP-1 gene expression by
inhibiting Sp1 and
AP-1 transcriptional activities in keratinocytes
Tonetti et al., J Steroid Biochem Mol Biol 2003
(Breast Neoplasms) :
Furthermore,
ERbeta , at least in the context of the MDA-MB-231 cellular milieu, does not
enhance AP-1 activity in the presence of antiestrogens
Schmitt et al., J Neurosci Res 1995
:
Using transient transfections in neuroblastoma NE-1-115 and COS-1 cells, we show that ligand activated
estrogen receptor ( HEGo )
represses the transcriptional activation by
c-Fos/c-Jun