◀ Back to RHOA
PXN — RHOA
Pathways - manually collected, often from reviews:
Text-mined interactions from Literome
Gakidis et al., J Cell Biol 2004
:
beta2 induced activation of Cdc42, Rac1, and
RhoA is defective in Vav1/3ko neutrophils, and phosphorylation of Pyk2,
paxillin , and Akt is also significantly
reduced
Lu et al., Mol Pharmacol 2006
:
Compared with docetaxel, laulimalide more potently caused a reduction in the constitutive levels ( i.e., in the absence of VEGF ) of phosphorylated
paxillin and more potently
inhibited the association of
RhoA with the alpha5beta1 integrin
Takemura et al., J Periodontol 2006
:
Phosphorylation of
paxillin and FAK by PDL cells was induced, and
RhoA activation also was
induced
Shafikhani et al., Cell cycle (Georgetown, Tex.) 2008
:
Our data demonstrate that the subcellular localization and the activity of
RhoA and citron K, which are essential for early stages of cytokinesis, are not
dependent on
paxillin , Crk or syntaxin-2
Deakin et al., PloS one 2012
:
Paxillin and Hic-5 interaction with vinculin is differentially
regulated by Rac1 and
RhoA
Zhang et al., J Biol Chem 2012
:
We assessed the mechanism by which
RhoA regulates actin dynamics and smooth muscle contraction by expressing the dominant negative mutants RhoA T19N and cdc42 T17N, and non-phosphorylatable paxillin Y118/31F and
paxillin dLD4 deletion mutants in SM tissues