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CD86 — RELA

Text-mined interactions from Literome

Suzuki et al., Immunology 2003 : These inhibitors interfered with CD86 gene transcription in the presence of activated NF-kappaB , whereas phosphorylated CREB was down-regulated in the reactions where these inhibitors were added to inhibit CD86 gene expression
Podojil et al., J Biol Chem 2004 : We showed that CD86 stimulation increased the nuclear localization of NF-kappaB1 ( p50 ) and phosphorylated RelA ( p65 ) and increased Oct-2 expression and binding to the 3'-IgH enhancer, in a protein kinase C-dependent manner
Zou et al., J Cell Physiol 2005 : We conclude that NF-kappaB signal plays a key role in LIGHT mediated upregulation of CD86 expression
Jatana et al., Journal of neuroinflammation 2006 : Furthermore, in vitro, BW-B 70C inhibited lipopolysaccharide (LPS) mediated nitric oxide production, iNOS induction and NF-kappaB activation in the BV2 microglial cell line
Yang et al., Biol Pharm Bull 2008 : Activation of NF-kappaB and ERK1/2 were necessary for CD80, CD86 and MHCII expression induced by semi-vioxanthin
Larangé et al., J Leukoc Biol 2009 : We found that upon TLR7 and TLR8 activation, JNK and NF-kappaB positively regulated the expression of CCR7, CD86 , CD83, and CD40 and the production of IL-6 and IL-12p40
Yin et al., Zhonghua Zhong Liu Za Zhi 2008 (Liver Neoplasms, Experimental) : H22-CD80/CD86/CD137L ( + ) induces higher NF-kappaB activity of the host T cells by synergistic action of CD28 and CD137, which may be one of the mechanisms of enhancement of the host CTL activity induced by co-expression of CD80, CD86 and CD137L genes