◀ Back to JUN

JUN — XIAP

Text-mined interactions from Literome

Dai et al., Mol Cell Biol 2005 (Leukemia) : Inhibition of NF-kappaB by pharmacological inhibitors or genetic strategies markedly potentiated apoptosis induced by HDACIs, and this was accompanied by enhanced reactive oxygen species ( ROS ) generation, downregulation of Mn-superoxide dismutase and XIAP , and c-Jun N-terminal kinase 1 ( JNK1 ) activation
Kaur et al., J Biol Chem 2005 (Carcinoma, Hepatocellular) : X-linked inhibitor of apoptosis (XIAP) inhibits c-Jun N-terminal kinase 1 ( JNK1 ) activation by transforming growth factor beta1 ( TGF-beta1 ) through ubiquitin mediated proteosomal degradation of the TGF-beta1 activated kinase 1 (TAK1)
Collett et al., Carcinogenesis 2006 : The p65/RelA subunit of NFkappaB may influence cell death, in part by activation of NFkappaB anti-apoptotic target genes including X-linked inhibitor of apoptosis (XIAP) , A20, bcl-xL and inhibition of sustained activation of c-Jun N-terminal kinase (JNK)
Lee et al., Arterioscler Thromb Vasc Biol 2008 : GADD45beta and XIAP , negative regulators of c-Jun N-terminal kinase (JNK) , were suppressed and JNK activity was increased
Nijboer et al., Stroke 2009 (Cerebral Infarction...) : We now show that TAT-NBD treatment increased HI-induced AP-1 activation concomitantly with reduced Gadd45beta, XIAP , and increased ( P ) -TAK1 expression