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ATP5O — EGF

Text-mined interactions from Literome

Falin et al., Am J Physiol Cell Physiol 2005 : The DBHQ mediated decrease in I ( sc ) was due to inhibition of basolateral Na ( + ) -K ( + ) -ATPase , but EGF- , ATP-, and PMA induced inhibition was most likely due to reduced apical sodium entry ( epithelial Na ( + ) channel activity )
Nitsche et al., Am J Physiol Gastrointest Liver Physiol 2007 : BMP-4 attenuated EGF mediated inhibition of H ( + ) /K ( + ) -ATPase gene expression and blocked EGF induction of both parietal cell morphological transformation and MAPK activation
Hughes et al., Mol Pharmacol 1991 : The Ca ( 2+ ) -ATPase inhibitor thapsigargin also activated Ca2+ entry, and neither epidermal growth factor nor the guanine nucleotide dependent protein linked receptor agonist bradykinin activated additional Ca2+ entry over that due to thapsigargin
Knowles et al., Arch Biochem Biophys 1990 (Carcinoma, Hepatocellular...) : The tyrosine kinase activity of the EGF receptor was essential for the induction of ectoCa2 ( + ) -ATPase , since enzyme induction was abolished by a tyrosine kinase inhibitor, genistein
Knowles et al., Biochem Biophys Res Commun 1985 (Carcinoma, Hepatocellular...) : The increased expression of the ectoCa2+-ATPase was absolutely dependent on EGF , but also required hydrocortisone and cholera toxin
Kaise et al., J Biol Chem 1995 : EGF in concentrations equivalent to those in plasma increased H+, K ( + ) -ATPase alpha-subunit mRNA levels ... Our studies indicate that EGF induces gastric H+, K ( + ) -ATPase alpha-subunit gene expression via an interaction between a specific ERE and a novel transcriptional factor and that this may be a physiologic mechanism by which EGF regulates acid secretion
Soler et al., Regul Pept 1994 : Effects of epidermal growth factor on gluconeogenesis and cellular redox state do not require Na+/H+ exchange or Na+/K ( + ) -ATPase activities ... Although Na+/H+ exchange and/or Na+/K ( + ) -ATPase are regulated by EGF , our results indicate that these activities are not required for the effects of EGF on gluconeogenesis and/or cytosolic and mitochondrial redox state
Borok et al., Am J Physiol 1996 : Net sodium flux and Na+, K+ -ATPase activity both increased approximately 50 % in the presence of EGF
Mehta et al., JPEN J Parenter Enteral Nutr 1997 : Effects of EGF on the kinetic parameters of sodium-glucose contransporter, thymidine transport, and on the activity of Na+/K ( + ) -ATPase were examined