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JUN — LEP

Text-mined interactions from Literome

Barkan et al., Endocrinology 1999 : We find that leptin induces c-Jun expression and attenuates the transcriptional activity of the glucocorticoid receptor ( GR ) in granulosa cells
Bendinelli et al., Mol Cell Endocrinol 2000 : Leptin activates Stat3, Stat1 and AP-1 in mouse adipose tissue ... AP-1 binding activity was also induced in adipose tissue by in vivo leptin treatment with a time course that suggests a post-transcriptional inductive mechanism
Fu et al., Cell 2005 : Partially antagonizing this inhibitory loop, leptin also upregulates AP-1 gene expression, which promotes cyclin D1 expression, osteoblast proliferation, and bone formation
Fu et al., Prog Brain Res 2006 : Indeed, we found that leptin dependent sympathetic signaling induces the expression of AP1 and circadian genes in bone and in osteoblasts with similar kinetics, and these two pathways play opposite roles in controlling c-myc expression
Wang et al., Gastroenterology 2009 (Disease Models, Animal...) : Leptin potentiated signal transducer and activator of transcription 3, AKT, and extracellular signal related kinase 1/2 phosphorylation in KCs and increased AP-1 and nuclear factor-kappaB DNA binding
Rucinski et al., Int J Mol Med 2010 : In comparison with intact rats, expression levels of ppGHREL, BAX, JUN-B and JUN-C genes were notably higher in regenerating adrenals, and neither GHREL nor OBS infusion affected these levels