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CRK — RNF19A

Text-mined interactions from Literome

Hale et al., J Immunol 1999 (Inflammation) : Immunokinase assays showed a strong activation of p38alpha and a lesser activation of p38delta in LPS stimulated macrophages
Wang et al., Exp Cell Res 2000 (MAP Kinase Signaling System) : Surprisingly, inhibition of p38 activity resulted in sustained enhancement of p38 phosphorylation and of its in vitro activity in the absence of the inhibitor, indicating up-regulation of the upstream components of the p38 pathway
Yamagishi et al., Eur J Biochem 2002 (Teratocarcinoma) : These findings indicated that the activation of p38 is an essential step for apoptosis in F9 cells and that Hsp105alpha enhances activation of p38 , release of cytochrome c and caspase activation
Duan et al., Am J Respir Crit Care Med 2005 (Asthma...) : Quantitative polymerase chain reaction analysis of bronchoalveolar lavage fluid cells and peribronchial lymph node cells showed that p38alpha-ASO significantly reduced p38alpha MAPK mRNA expression
Kim et al., J Immunol 2005 (Infection...) : Furthermore, a synthetic p38 catalytic-site inhibitor blocked tachyzoite induced p38alpha MAPK phosphorylation
Peng et al., Circulation 2005 (Cardiomyopathies) : NADH oxidase activation by LPS increased ERK1/2 and p38 phosphorylation, and inhibition of ERK1/2 and p38 phosphorylation blocked the effect of NADH oxidase on TNF-alpha expression
Koizumi et al., Anticancer Res 2005 (Adenocarcinoma...) : Overexpression of a dominant negative p38 mutant in cells resulted in the reduction of gemcitabine induced p38 MAPK activation and apoptosis, and increases in clonogenic survival
Rushworth et al., Biochem Biophys Res Commun 2006 : Furthermore, a p38 MAP kinase inhibitor reduced GCLM and HO-1 expression and rottlerin inhibited curcumin induced p38 phosphorylation
Berenson et al., J Immunol 2006 : These results suggest that the activity of p38alpha in Th1 cells is relatively restricted to acting in one of two alternative pathways ( i.e., cytokine induced ) that can induce the production of IFN-gamma in differentiated Th1 cells, but that p38alpha is not required for the process of Th1 commitment and development itself
Naïmi et al., Endocrinology 2007 (MAP Kinase Signaling System) : In contrast, Foxo1-ADA increases p38 activity, and p38 is required for effects of Foxo1 on PKB, at least in part
Schett et al., Ann Rheum Dis 2008 (Arthritis, Rheumatoid...) : This review examines the function of one key signal transduction pathway of inflammation -- the p38 mitogen activated protein kinases ( p38MAPK )
Lee et al., J Biochem 2010 (Leukemia, T-Cell) : Overexpression of p38alpha ( AF ) or p38beta ( AF ) inhibited IFN-alpha mediated p38 kinase activity and growth inhibition in Jurkat cells
Brighton et al., Br J Pharmacol 2011 (MAP Kinase Signaling System) : Conversely, depletion of arrestin3 enhanced p38 signals to each agonist, whilst arrestin2 suppression increased oxytocin-, but not histamine induced p38 MAPK responses
Huong et al., The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology 2012 : Furthermore, the activation of p38 was synergistically induced by GDB plus IFN-?, but SB203580 ( a p38 inhibitor ) inhibited GDB plus IFN-? induced p38 activation
Yuasa et al., J Biol Chem 1998 : We show that RIP activates both p38 and SAPK ; and that TRAF2 activation of p38 requires RIP