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FGF19 — TCF21

Text-mined interactions from Literome

Xu et al., Dev Dyn 2000 : We have previously demonstrated that the transcription factor , AP-1 ( c-jun/c-fos heterodimer ), mediates fibroblast growth factor ( FGF ) signaling during mesoderm induction in Xenopus embryo
Cross et al., Placenta 2003 : Differentiation towards syncytiotrophoblast as well as the initiation of chorioallantoic ( villous ) morphogenesis is regulated by the Gcm1 transcription factor , and subsequent labyrinth development is dependent on Wnt, HGF and FGF signalling
Katoh et al., Cancer Biol Ther 2007 (Helicobacter Infections...) : WNT-beta-catenin-TCF/LEF signaling induces upregulation of MYC, CCND1, WISP1, FGF20 , JAG1 and DKK1 genes
Saarimäki-Vire et al., J Neurosci 2007 : Our results suggest a SoxB1 family member, Sox3, as a potential FGF induced transcription factor promoting progenitor renewal
Pai et al., Cancer Res 2008 (Colonic Neoplasms) : Using HCT116 paired beta-catenin knockout cells, we show that FGF19 induces TCF/LEF reporter activity in parental ( WT/Delta45 ) and in WT/ -- but not in mutant ( -/Delta45 ) cells, and that inhibition of endogenous FGF19 reduces this reporter activity, indicating that wild-type beta-catenin is accessible for modulation
Mazzoni et al., Nat Neurosci 2013 : Wnt and fibroblast growth factor ( FGF ) signals induce expression of the Cdx2 transcription factor that binds and clears H3K27me3 from the Hox1-Hox9 chromatin domains, leading to specification of brachial or thoracic spinal identity
Samakovlis et al., Development 1996 : We further show that the breathless FGF receptor, a tracheal gene required for primary branching, is also required to activate expression of markers involved in secondary branching and that the pointed ETS-domain transcription factor is required for secondary branching and also to activate expression of terminal branch markers