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IL16 — PTBP2

Text-mined interactions from Literome

Manome et al., Immunology 1999 (Dermatitis, Contact) : Finally, we examined the effects of these chemicals on CD86 expression by three different macrophage subsets and DCs induced from the cultures of human peripheral blood monocytes in the presence of macrophage colony stimulating factor ( M-CSF ), M-CSF + interleukin-4 (IL-4), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and GM-CSF + IL-4, respectively
Liu et al., Chin J Cancer 2003 (Breast Neoplasms) : DCs were stimulated by granulocyte/macrophage colony stimulating factor ( GM-CSF ), interleukin-4(IL-4) , and tumor antigen
Reich et al., Exp Dermatol 2004 (Dermatitis, Atopic...) : Maturation, as determined by up-regulation of CD83 and CD86 surface expression, and production of IL-16 , but not production of IL-10 and IL-12p40 was impaired in day 8 DCs derived from AD patients compared to those from healthy donors ... Stimulation of day 8 DCs from AD patients with TNF-alpha and IL-1beta enhanced the expression of CD83 and CD86 and restored the production of IL-16
Hua et al., Nan Fang Yi Ke Da Xue Xue Bao 2006 : The DCs were derived from healthy human peripheral blood monocytes in the presence of granulocyte-macrophage colony stimulating factor, interleukin (IL)-4 and tumor necrosis factor (TNF) alpha
Byun et al., Biochem Biophys Res Commun 2012 (Inflammation) : In addition, EGCG treated DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a, interleukin [ IL]-1ß, and IL-6 ) and activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and nuclear factor ?B ( NF-?B ) p65 translocation through 67LR
Khanna et al., Transplantation 1998 : In addition, however, a link was observed between the beneficial effect of donor BM and comparatively large numbers of donor major histocompatibility complex class II ( IAb+ ) -positive cells in recipients ' spleens, and in cultures of granulocyte-macrophage colony stimulating factor + interleukin-4 stimulated DCs from recipients ' BM