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IL2 — PDCD11

Text-mined interactions from Literome

Di Renzo et al., Arch Dermatol Res 2003 (Chronic Disease...) : This enhanced programmed cell death could not be prevented by IL-2
Matsui et al., Intensive Care Med 2012 : The production of tumor necrosis factor-alpha ( TNF-a ), interleukin-10 (IL-10), and NO and the activation of NF-?B p65 were reduced by hypothermia, but augmented by hyperthermia at 3-6, 24-48, 48, and 0.5 h, post-treatment initiation, respectively
Byun et al., Biochem Biophys Res Commun 2012 (Inflammation) : In addition, EGCG treated DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a, interleukin [ IL]-1ß, and IL-6 ) and activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and nuclear factor ?B ( NF-?B ) p65 translocation through 67LR
Wilson et al., Nature 1994 : Interleukin-1 beta converting enzyme ( ICE ) processes an inactive precursor to the proinflammatory cytokine, interleukin-1 beta, and may regulate programmed cell death in neuronal cells
Huang et al., Int J Hematol 1993 (Leukemia, Lymphocytic, Chronic, B-Cell...) : Interleukin-2 prevents programmed cell death in chronic lymphocytic leukemia cells
Deng et al., Proc Natl Acad Sci U S A 1993 : Absence of interleukin 2 (IL-2) from IL-2 dependent cells, such as the cytotoxic T-cell line CTLL2, causes DNA fragmentation and programmed cell death ( apoptosis )
Tsuda et al., Jpn J Cancer Res 1993 (Leukemia, T-Cell) : Interleukin-2 prevents programmed cell death in adult T-cell leukemia cells