Gene interactions and pathways from curated databases and text-mining

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IRF6 — LTF

Text-mined interactions from Literome

Mitsuhashi et al., Acta Obstet Gynecol Scand 2000 (Disease Models, Animal...) : Effect of lactoferrin on lipopolysaccharide (LPS) induced preterm delivery in mice ... The present study attempted to confirm the preventive effect of lactoferrin on lipopolysaccharide (LPS) induced preterm delivery in mice
Palma et al., Infect Immun 1992 (Candidiasis) : LPS activation caused augmented release of lactoferrin , an iron binding protein which itself could inhibit the growth of C. albicans in vitro
Al-Haddawi et al., Vet Immunol Immunopathol 2007 (Bovine Virus Diarrhea-Mucosal Disease...) : These data indicate that infection with a type 2 BVDV isolate inhibits the LPS induced upregulation of TAP mRNA expression and lactoferrin secretion by tracheal epithelial cells, suggesting a novel mechanism by which this virus abrogates respiratory innate immune responses and predisposes to bacterial pneumonia in cattle
Shen et al., Clin Exp Immunol 2007 : We observed that incubation of neutrophils in vitro with TGF-beta1 at 1 pg/ml significantly reduced their secretion of lactoferrin in response to lipopolysaccharide (LPS)
Moreno-Navarrete et al., J Clin Endocrinol Metab 2009 (Chronic Disease...) : In ex vivo experiments, we found a significant decrease in LPS induced lactoferrin release from neutrophils in subjects with type 2 diabetes ... IL-6 coincubation decreased LPS induced lactoferrin release in NGT subjects ( P < 0.001 )
Ellison et al., Infect Immun 1988 : Initial studies in barbital-acetate buffer showed that EDTA and lactoferrin cause significant release of LPS from all three strains
Koivuranta-Vaara et al., Infect Immun 1987 : LPS induced release of lactoferrin from PMN was augmented significantly when cell suspensions were supplemented with additional monocytes and lymphocytes ... Thus, LPS induced noncytotoxic release of lactoferrin from human PMN suspended in serum-free buffer is mediated, at least in part, by tumor necrosis factor alpha derived from contaminating monocytes