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CRK — MMP9

Text-mined interactions from Literome

Xue et al., Arthritis Rheum 2007 (Arthritis, Rheumatoid...) : Furthermore, APC directly suppressed the production of tumor necrosis factor (TNF) and the activation of NF-kappaB and MAP kinase p38, and inhibitors of NF-kappaB or p38 reduced the production of MMP-9, suggesting that APC inhibits MMP-9 by blocking TNF, NF-kappaB, and p38
Mühlen et al., Virus Res 2009 (Carcinoma...) : AP-1 and ERK1 but not p38 nor JNK is required for CRPV early protein 2-dependent MMP-9 promoter activation in rabbit epithelial cells
Lee et al., Inflammation 2011 (Breast Neoplasms...) : These signaling molecules and additional p38 ( MAPK ) were also involved in macrophage induced MMP-9 expression
Turner et al., Matrix Biol 2010 : Pharmacological inhibitors of p38-a/ß ( SB203580 ) or p38-a/ß/?/d ( BIRB-0796 ) reduced MMP-3 and ADAMTS1 mRNA expression, but neither inhibitor affected MMP-9 levels
Ling et al., Br J Pharmacol 2010 (Breast Neoplasms...) : By using specific inhibitors, it was demonstrated that ERK and NF-?B signalling, but not JNK and p38 signalling, were involved in PMA stimulated MMP-9 activation
Deraz et al., PloS one 2011 (Carcinoma, Squamous Cell...) : Interestingly, MMP-10 overexpression induced the decreased p38 activity and MMP-10 knockdown induced the increased p38 activity
Heijink et al., Thorax 2013 (Pulmonary Disease, Chronic Obstructive) : Exogenously added WNT-4 significantly increased CXCL8/IL-8, IL-6, CCL5/RANTES, CCL2/MCP-1 and vascular endothelial growth factor ( VEGF ) secretion in 16HBE, but did not affect the canonical WNT target genes MMP-2, MMP-9 , fibronectin, ß-catenin, Dickkopf and axin-2, and induced activation of the non-canonical signalling molecule p38