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NGF — TCF19

Text-mined interactions from Literome

Khursigara et al., J Neurosci 2001 : The introduction of RIP2 into Schwann cells deficient in RIP2 conferred NGF dependent nuclear transcription factor-kappaB ( NF-kappaB ) activity and decreased the cell death induced by NGF
Cosgaya et al., J Neurochem 2001 : Nerve growth factor (NGF) regulates the activity of the transcription factor NF-kappaB ( nuclear factor-kappaB ) through its low affinity receptor, p75
Oh et al., J Toxicol Environ Health 2004 : Since MAP kinase and transcription factors have been implicated in cell differentiation of neuronal cells, and our previous study demonstrated that p38 MAP kinase and AP-1 are activated during PC 12 cell differentiation, the effect of OTA on NGF induced p38 MAP kinase and transcription factor activation was examined
Ahamed et al., J Immunol 2004 : Furthermore, C3a enhances NGF induced transcription factor activation and chemokine production via a G protein mediated pathway that does not involve TrkA phosphorylation
Kimura et al., J Cell Biol 1992 : In addition, both SDGF and NGF induce the transcription factor , NGFI-A
McCauslin et al., J Biol Chem 2006 (Glioma) : We previously showed that the transcription factor CCAAT/enhancer binding protein delta ( C/EBPdelta ) contributes to NGF gene regulation
Su et al., J Neurosci Res 2007 : We conclude that preferential activation of transcription factor binding at this AP-1 site mediates preferential NGF activation of the UPAR gene
Takei et al., Mol Biol Cell 2008 (Neuroblastoma...) : We find that NGF can induce TNF-alpha synthesis through the nuclear factor-kappaB transcription factor
Leugers et al., J Biol Chem 2010 (MAP Kinase Signaling System) : In addition, we demonstrate that Tau potentiates AP-1 transcription factor activation in response to nerve growth factor (NGF)
Li et al., PloS one 2011 (Atrophy) : Egr3 is a transcription factor regulated by NGF signaling in sympathetic neurons that is essential for normal SNS development
deSouza et al., J Mol Neurosci 1997 : On the other hand, expression of a mutated form of this transcription factor lacking the DNA binding domain attenuated NGF responsiveness, without affecting basal levels of expression
Furukawa et al., J Neurosci Res 1998 : Presenilin-1 mutation alters NGF induced neurite outgrowth, calcium homeostasis, and transcription factor ( AP-1 ) activation in PC12 cells