◀ Back to CSF3

CSF3 — NOS2

Text-mined interactions from Literome

Deetjen et al., Immunopharmacology 1999 : Qualitative and quantitative analyses of iNOS cDNA revealed that G-CSF significantly reduced interferon-gamma/lipopolysaccharide ( IFN-gamma/LPS ) dependent iNOS gene expression ( P < 0.05 ) following 6, 18, 24, and 48 h incubation periods
Stephan et al., Int J Tissue React 1999 : However, both IL-1 beta and G-CSF caused a significant increase ( p < 0.05 ) in nitrite levels at 48 h. NG-L-arginine-methyl-ester was used to inhibit nitrite production induced by G-CSF and this implicated nitric oxide synthase activity
Hierholzer et al., Am J Physiol Gastrointest Liver Physiol 2004 (Enteritis...) : Selective iNOS inhibition in shocked animals reduced the binding activity of NF-kappaB and Stat3 and reduced mRNA levels of ICAM-1, IL-6, and G-CSF
Wai et al., Surgery 2006 (Adenocarcinoma...) : In our system of CT26-RAW264.7 coculture, we conclude that inhibition of OPN synthesis in CT26 results in G-CSF mediated induction of macrophage iNOS expression with resultant inhibition of CT26 proliferation via increased apoptosis
Oppeltz et al., Cytokine 2012 (Burns) : Inhibition of iNOS activity in WT mice with L-Nil suppressed burn wound levels of IL-1ß, G-CSF , and MIP-1a, whereas IL-6, TNF-a, KC, MCP-1 and MIP-1ß were unaffected
Squadrito et al., Br J Pharmacol 1997 (Mesenteric Vascular Occlusion...) : Finally rh G-CSF potently inhibited the activity of inducible nitric oxide synthase in peritoneal macrophages activated with endotoxin
Squadrito et al., Int J Microcirc Clin Exp 1997 (Acute Disease...) : Finally, rh G-CSF inhibited the activity of inducible nitric oxide synthase in peritoneal macrophages activated with endotoxin
Hierholzer et al., J Exp Med 1998 (Inflammation...) : Using the iNOS inhibitor N6- ( iminoethyl ) -L-lysine or iNOS knockout mice we found that the activation of the transcriptional factors nuclear factor kappaB and signal transducer and activator of transcription 3 and increases in IL-6 and G-CSF messenger RNA levels in the lungs and livers measured 4 h after resuscitation from hemorrhagic shock were iNOS dependent
Hoffmann et al., Eur J Pharmacol 1998 : Granulocyte-colony stimulating factor inhibits inducible nitric oxide synthase gene expression in pulmonary epithelial cells in vitro ... We show that G-CSF suppresses interferon-gamma/tumor necrosis factor-alpha (TNF-alpha) induced inducible nitric oxide synthase gene expression detected as inducible nitric oxide synthase cDNA ( cDNA concentration was 633 +/- 38 amol/microg total RNA following 24 h incubation with 100 U/ml interferon-gamma/500 U/ml TNF-alpha, and 440 +/- 14 amol/microg total RNA following 24 h incubation with 250 U/ml G-CSF +/- 100 U/ml interferon-gamma/500 U/ml TNF-alpha, respectively )