◀ Back to CALM3

CALM3 — PI3

Text-mined interactions from Literome

Vergne et al., J Exp Med 2003 (Calcium Signaling) : The interference of the trafficking toxin LAM with the calmodulin dependent production of PI3P described here ensures long-term M. tuberculosis residence in vacuoles sequestered away from the bactericidal and antigen processing organelles in infected macrophages
Pérez-García et al., J Biol Chem 2004 : We conclude that changes of [ Ca2+ ] i, induced by GDNF, promote neuronal survival through a mechanism that involves a direct regulation of PI 3-kinase activation by CaM thus suggesting a central role for Ca2+ and CaM in the signaling cascade for neuronal survival mediated by neurotrophic factors
Thors et al., FEBS Lett 2004 : Inhibition of the Ca2+/calmodulin dependent protein kinase II or phosphoinositide 3-kinase (PI3K) had no effect
Siarey et al., J Neurochem 2006 (Down Syndrome) : Here we report that calcium/calmodulin dependent protein kinase II ( CaMKII ), phosphatidylinositol 3-kinase (PI3K)/Akt , extracellular signal regulated kinase ( ERK ), protein kinase A (PKA) and protein kinase C ( PKC ), all of which have been shown to be involved in synaptic plasticity, are altered in the Ts65Dn hippocampus
Xu et al., Hippocampus 2007 (Brain Infarction...) : Subtoxic N-methyl-D-aspartate delayed neuronal death in ischemic brain injury through TrkB receptor- and calmodulin mediated PI-3K/Akt pathway activation
Lasbury et al., Infect Immun 2009 (Pneumonia, Pneumocystis) : Experiments to determine the control of GM-CSF and PI-3K by calmodulin in Amo showed that GM-CSF expression and PI-3K activation could not be induced when calmodulin was inhibited
Yamagata et al., Microvasc Res 2012 (Atherosclerosis) : IL-1ß decreased the expression of Ca2+/calmodulin dependent kinase II ( CaMKII ), eNOS, PON-1, phosphatidylinositol 3-kinase (PI3K) , PSD-95/Dlg/ZO-1 ( PZK1 ), and liver kinase B1 (LKB1)
Yamagata et al., Microvasc Res 2013 (Arteriosclerosis) : The expression of genes associated with HDL effects such as Ca ( 2+ ) /calmodulin dependent kinase II ( CaMKKII ), liver kinase B ( LKD1 ), PSD-95/Dlg/ZO-1 kinase 1 (PDZK1), phosphatidylinositol 3-kinase (PI3K) , intercellular adhesion molecule-1 ( ICAM-1 ), monocyte chemotactic protein-1 (MCP-1), and endothelial nitric oxide synthase (eNOS) was examined by RT-PCR, and ICAM-1 protein expression was evaluated by western blot ( WB )