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PLAUR — TNF

Text-mined interactions from Literome

Niiya et al., Cancer Chemother Pharmacol 2003 (Carcinoma, Non-Small-Cell Lung...) : Furthermore, the cells did not express receptors including types I and II TNF-alpha receptors, uPA receptor (uPAR), C-x-C-chemokine receptor-1 (CXCR-1), or C-C-chemokine receptor-2, corresponding to TNF-alpha , uPA, IL-8 and MCP-1, respectively, that were induced by doxorubicin in the cells, although SBC-7 cells expressed uPAR , and EBC-1 cells expressed CXCR-1
Iwamoto et al., Dig Dis Sci 2003 (Stomach Ulcer) : To clarify the role of human gastric fibroblasts in acute inflammatory conditions such as gastric ulcer, the effects of interleukin (IL)-1beta and tumor necrosis factor (TNF)-alpha on the expression of uPA and uPAR , which were suggested to be associated with tissue remodeling, in gastric fibroblasts were investigated
Sitrin et al., Blood 1994 : By contrast, TNF alpha induced a 2.5-fold increase in the level of uPAR protein released into conditioned medium ( compared with unstimulated cells ), whereas IFN gamma had no effect
Koolwijk et al., J Cell Biol 1996 (Neovascularization, Pathologic) : bFGF and VEGF165 enhanced of the u-PAR by 72 and 46 %, but TNFalpha itself also increased u-PAR in hMVEC by 30 %