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PRKAA1 — VCAM1

Text-mined interactions from Literome

Cacicedo et al., Biochem Biophys Res Commun 2004 : The increases in NF-kappaB activation and VCAM-1 expression caused by palmitate were markedly inhibited by co-incubation with the AMPK activator AICAR and, where studied, by expression of a constitutively active AMPK
Suzuki et al., Am J Hypertens 2008 (Diabetes Mellitus, Experimental...) : Activation of AMPK by cilostazol was through signaling pathway independent of cyclic AMP and caused phosphorylation of endothelial nitric oxide synthase (eNOS), leading to increased production of nitric oxide ( NO ), while inhibiting cytokine induced nuclear factor-kappaB (NF-kappaB) activation, leading to suppression of VCAM-1 gene expression
Zhang et al., Arterioscler Thromb Vasc Biol 2011 (Inflammation) : 5-Aminoimidazole-4-carboxamide-1-ß-D-ribonucleotide ( AICAR ) treatment or overexpression of constitutively active AMPK markedly reduced human monocytic human acute monocytic leukemia cell line-1 cell adhesion and the expression of vascular cell adhesion molecule-1 in tumor necrosis factor-a activated human aortic endothelial cells