Gene interactions and pathways from curated databases and text-mining

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PTK2 — TP53

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Pierce et al., Oncogene 2000 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : We conclude that prolonged Bcr-Abl PTK activity within multipotent cells results in a reduction of p53 that drives a delayed and abnormal differentiation
Zhang et al., Pharmacology 2002 (Stomach Neoplasms) : Further studies showed that As ( 2 ) O ( 3 ) could regulate protein tyrosine kinase activity, protein tyrosine phosphorylation, and Bcl-2 protein and upregulate p53 protein
Corey et al., EMBO J 1993 : Here we show that in human myeloid derived cells GM-CSF can stimulate ; ( i ) the accumulation of PtdIns ( 3,4,5 ) P3 ; ( ii ) increases in p53/p56lyn and p62c-yes directed protein tyrosine kinase activities in anti-lyn and anti-c-yes antibody directed immunoprecipitates, respectively and ; ( iii ) increases in phosphoinositide 3OH-kinase activity in antiphosphotyrosine, anti-p53/p56lyn and anti-p62c-yes antibody directed immunoprecipitates