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IL10 — SEA

Text-mined interactions from Literome

Velupillai et al., Hum Immunol 2000 (Schistosomiasis mansoni) : Soluble egg antigens (SEA) also induced IL-10 production by PBMC from infected patients ... Interestingly, the SEA induced IL-10 production was remarkably inhibited by pretreatment of PBMC with free ligands of LNFPIII ( monovalent form )
Farah et al., Scand J Immunol 2000 (Granuloma...) : Group B animals had low SEA induced IFN-gamma levels and elevated IL-5 levels, although these were lower than group C. SEA induced IL-10 was low in both groups A and B as compared to group C ( P < 0.01 )
Hegardt et al., Cell Immunol 2000 (Disease Progression...) : Plastic adherent spleen cells from tumor bearing rats suppressed the SEA induced proliferative response and the production of IFN-gamma and IL-10 by nonadherent spleen cells from tumor-free rats
Tangsinmankong et al., Cytokine 2000 : By contrast, SEA induced IL-10 production as well as mRNA expression using T lymphocytes but not with monocytes or B lymphocytes
King et al., Parasite Immunol 2001 (Schistosomiasis mansoni) : By contrast, CD4+ splenocytes produced comparatively little cytokine release by endogenous antigen, whereas exogenous SEA strongly induced IL-4, IL-5, IL-10 and IFN-gamma production and lymphocyte proliferation that correlated poorly with the dynamics of granulomatous inflammation
Tangsinmankong et al., Immunol Lett 2002 : Recently, we demonstrated that HIV-1 Nef induces IL-10 production in monocytes and that staphylococcal enterotoxin A (SEA) induces IL-10 production in T-lymphocytes ... Conversely, we show that CsA and FK506 completely inhibit SEA induced IL-10 protein production and mRNA expression
Ramos et al., Mem Inst Oswaldo Cruz 2006 (Disease Models, Animal...) : Interleukins, IL-4 and IL-10 responses to soluble egg antigens (SEA) 90 days after infection, were determined
Huang et al., Acta Biochim Biophys Sin (Shanghai) 2007 : Repeated SEA stimulation led to down-regulation of interleukin (IL)-2, and high levels of IL-10
Rutitzky et al., J Immunol 2008 (Liver Diseases, Parasitic...) : We now show that mice lacking the IL-23-specific subunit p19 are impaired in developing severe immunopathology following immunization with SEA/CFA, which is associated with a marked drop of IL-17 in the granulomas, but not in the draining mesenteric lymph nodes, and with a markedly suppressed SEA-specific IFN-gamma response regulated by a striking increase in IL-10
Correale et al., J Immunol 2009 (Multiple Sclerosis...) : SEA suppressed the LPS induced DCs production of IL-1beta, IL-6, IL-12, and TNF-alpha and enhanced TGF-beta as well as IL-10 production
Ferreira et al., Mem Inst Oswaldo Cruz 2012 (Chronic Disease...) : We demonstrate that stimulation with SEA induced prominent lymphocyte proliferation and high levels of tumour necrosis factor (TNF)-a, interleukin (IL)-4 and IL-10 secretion in both OST and non infected individuals ( NI )
da Silva et al., TheScientificWorldJournal 2012 (Schistosomiasis mansoni) : Histopathological analysis of liver tissue showed changes in morphological aspects of the hepatic parenchyma with decrease exudative-productive hepatic granuloma stages, although we found no significant differences in IFN-?, IL-4, IL-10 , or NO production in response to the specific antigen SEA
Boghdadi et al., Parasitol Res 2013 (Liver Cirrhosis...) : Levels of interferon-? ( IFN-? ), interleukin 4 (IL-4), IL-10 , and IL-13 were measured by ELISA in the supernatants of cultured peripheral blood mononuclear cells ( PBMC ) in response to schistosoma egg soluble antigen (SEA) in the presence and absence of R848
Tang et al., PloS one 2012 : The production of IL-10 and IL-6 in RAW 264.7 cells, induced by SEA , was responsible for mediating the down-regulation of MHC class II ... Our findings suggest that in RAW 264.7 cells ( 1 ) IFN-? provides a condition for lower concentrations of SEA to attenuate MHC class II expression ; ( 2 ) SEA attenuated IFN-? induced MHC class II expression and the IL-10 and IL-6 production is mediated at least partly by the interaction of SEA with TLR4 ; and ( 3 ) SEA attenuated IFN-? induced MHC class II expression at the transcriptional level
King et al., J Immunol 1996 (Chronic Disease...) : SWAP or SEA triggered IL-10 release from PBMC of both patients and healthy individuals ; however, CD4+ cells were a significant source of IL-10 only in infected subjects
Hobeika et al., Biochem Biophys Res Commun 1996 : A synthetic peptide of this region, SEA ( 121-149 ), blocks SEA binding to class II MHC molecules and induces interleukin-1 and tumor necrosis factor production in monocytes
Sundstedt et al., J Immunol 1997 : Depletion of T cells with anti-CD4 or anti-CD8 mAb indicated that CD4+ T cells were crucial for SEA induced IL-10 production
Bosshardt et al., Parasite Immunol 1997 (Chronic Disease...) : In contrast, while SEA stimulated IL-10 production was again highest with cells from mice with acute infections, spleen cells from mice with MSS produced significantly more IL-10 than did those from mice with HSS