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CA2 — SLC25A3

Text-mined interactions from Literome

Eriksson et al., Am J Physiol 1999 : Triggering of the permeability transition pore (PTP) in isolated mitochondria causes release of matrix Ca2+, ions, and metabolites, and it has been proposed that the PTP mediates mitochondrial Ca2+ release in intact cells
Huang et al., Mol Cell Biochem 2001 : The dependence of Ca2+ induced permeability transition pore (PTP) opening and cytochrome c ( Cyt. c ) release from mitochondria on mitochondria energetic status was investigated
Hajnóczky et al., Alcohol Clin Exp Res 2005 (Calcium Signaling...) : The authors ' results show that prolonged exposure of cultured cardiac cells to ethanol ( 35 mM for 48 hr ) promotes Ca2+ induced activation of the mitochondrial permeability transition pore (PTP)
Akopova et al., Ukr Biokhim Zh 2004 (Ion Channel Gating) : It is shown that mitochondria of rat myocardium exhibit high sensitivity to Ca2+ induced permeability transition pore (PTP) during an hour after their isolation
Pokhilko et al., J Theor Biol 2006 (Calcium Signaling) : Higher levels of Ca2+ ( > 160 nmol Ca2+/mg protein ) result in a permanently open PTP , membrane depolarization and loss of small ions from the matrix
Azarashvili et al., Cell Calcium 2007 : Here, we studied whether PBR is involved in Ca2+ induced permeability transition pore (PTP) opening in isolated rat brain mitochondria ( RBM )
Krestinina et al., Biochemistry (Mosc) 2009 : The effect of nanomolar concentrations of PBR/TSPO ligands -- Ro 5-4864, PK11195, and PPIX -- on Ca2+ induced permeability transition pore (PTP) opening in isolated rat brain mitochondria was investigated