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SMAD3 — STAT3

Text-mined interactions from Literome

Fukuda et al., Mol Cell Biol 2007 : Potentiation of astrogliogenesis by STAT3 mediated activation of bone morphogenetic protein-Smad signaling in neural stem cells
Park et al., J Invest Dermatol 2008 (Keloid) : The level of Smad2/3 , signal transducer and activator of transcription-3 ( STAT3 ), and p38 phosphorylation is more enhanced in KFs, and EGCG inhibited phosphorylation of phosphatidylinositol-3-kinase (PI3K), extracellular signal regulated protein kinase 1/2 ( ERK1/2 ), and STAT3 ( Tyr705 and Ser727 )
Bain et al., PloS one 2010 (Hypoxia-Ischemia, Brain) : TGFbeta1 induced Smad 2/3 phosphorylation and the combination of EGF, LIF and TGFbeta1 synergistically increased STAT3 phosphorylation over single or double cytokine combinations
Meisel et al., Immunity 2013 (Encephalomyelitis, Autoimmune, Experimental) : Furthermore, PKCa-deficient ( Prkca ( -/- ) ) cells demonstrated a defect in SMAD dependent IL-2 suppression, as well as decreased STAT3 DNA binding within the Il17a promoter